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Neuron|September 1, 2005
Neuregulin-1 type III determines the ensheathment fate of axonsCarla Taveggia, George Zanazzi, Ashley Petrylak, et al.Elife|March 24, 2016
PI(3,5)P2 biosynthesis regulates oligodendrocyte differentiation by intrinsic and extrinsic mechanismsYevgeniya A Mironova, Guy M Lenk, Jing-Ping Lin, et al.Neurobiology of Disease|January 26, 2016
Single amino acid deletion in transmembrane segment D4S6 of sodium channel Scn8a (Nav1.6) in a mouse mutant with a chronic movement disorderJulie M Jones, Louise Dionne, James Dell'Orco, et al.Proceedings of the National Academy of Sciences of the United States of America|December 14, 2002
Reduced sodium channel density, altered voltage dependence of inactivation, and increased susceptibility to seizures in mice lacking sodium channel beta 2-subunitsChunling Chen, Vandana Bharucha, Yuan Chen, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 23, 2004
Mice lacking sodium channel beta1 subunits display defects in neuronal excitability, sodium channel expression, and nodal architectureChunling Chen, Ruth E Westenbroek, Xiaorong Xu, et al.Science Translational Medicine|October 8, 2025
Deletion of murine Sarm1 results in a microenvironment that delays peripheral nerve regeneration after injuryLigia B Schmitd, Hannah Hafner, Ayobami Ward, et al.Biorxiv : the Preprint Server for Biology|March 18, 2024
Sarm1 is not necessary for activation of neuron-intrinsic growth programs yet required for the Schwann cell repair response and peripheral nerve regenerationLigia B Schmitd, Hannah Hafner, Ayobami Ward, et al.Pageof 3