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Journal of Neuroinflammation|May 28, 2013
The neonatal CNS is not conducive for encephalitogenic Th1 T cells and B cells during experimental autoimmune encephalomyelitisPetra D Cravens, Bernd C Kieseier, Rehana Hussain, et al.
Annals of Neurology|May 4, 2010
Silencing Nogo-A promotes functional recovery in demyelinating diseaseYuhong Yang, Yue Liu, Ping Wei, et al.
Archives of Neurology|October 15, 2008
Decrease in the numbers of dendritic cells and CD4+ T cells in cerebral perivascular spaces due to natalizumabMaria del Pilar Martin, Petra D Cravens, Ryan Winger, et al.
Archives of Neurology|October 13, 2006
Altered CD4+/CD8+ T-cell ratios in cerebrospinal fluid of natalizumab-treated patients with multiple sclerosisOlaf Stüve, Christina M Marra, Amit Bar-Or, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 31, 2021
CD11c+CD88+CD317+ myeloid cells are critical mediators of persistent CNS autoimmunityNavid Manouchehri, Rehana Z Hussain, Petra D Cravens, et al.
Archives of Neurology|August 12, 2009
Depletion of B lymphocytes from cerebral perivascular spaces by rituximabMaria del Pilar Martin, Petra D Cravens, Ryan Winger, et al.
Archives of Neurology|February 14, 2007
Potential risk of progressive multifocal leukoencephalopathy with natalizumab therapy: possible interventionsOlaf Stüve, Christina M Marra, Petra D Cravens, et al.
Brain : a Journal of Neurology|February 11, 2010
Pharmacological prion protein silencing accelerates central nervous system autoimmune disease via T cell receptor signallingWei Hu, Stefan Nessler, Bernhard Hemmer, et al.
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