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Der Hautarzt; Zeitschrift Fur Dermatologie, Venerologie, Und Verwandte Gebiete|January 23, 2020
[Extended understanding of pathogenesis and treatment of contact allergy]Philipp R Esser, Stefan F MartinHandbook of Experimental Pharmacology|June 26, 2021
Innate Immune Mechanisms in Contact DermatitisStefan F Martin, Philipp R EsserCurrent Allergy and Asthma Reports|November 13, 2017
Pathomechanisms of Contact SensitizationPhilipp R Esser, Stefan F MartinExperientia Supplementum (2012)|November 12, 2013
Correlation of contact sensitizer potency with T cell frequency and TCR repertoire diversityPhilipp R Esser, Ian Kimber, Stefan F MartinEuropean Journal of Immunology|April 4, 2023
Endoplasmic reticulum stress and the inflammatory response in allergic contact dermatitisPhilipp R Esser, Michael Huber, Stefan F MartinPlanta Medica|March 17, 2019
Plant Allergen-Induced Contact DermatitisPhilipp R Esser, Sabine Mueller, Stefan F MartinEMBO Reports|October 13, 2012
Metal allergens nickel and cobalt facilitate TLR4 homodimerization independently of MD2Badrinarayanan Raghavan, Stefan F Martin, Philipp R Esser, et al.Contact Dermatitis|July 4, 2021
Feeding of a fat-enriched diet causes the loss of resistance to contact hypersensitivityAnne-Catherine Rühl-Muth, Mareike D Maler, Philipp R Esser, et al.Rejuvenation Research|September 26, 2012
Luteolin prevents solar radiation-induced matrix metalloproteinase-1 activation in human fibroblasts: a role for p38 mitogen-activated protein kinase and interleukin-20 released from keratinocytesUte Wölfle, Anja Heinemann, Philipp R Esser, et al.Free Radical Biology & Medicine|February 2, 2011
UVB-induced DNA damage, generation of reactive oxygen species, and inflammation are effectively attenuated by the flavonoid luteolin in vitro and in vivoUte Wölfle, Philipp R Esser, Birgit Simon-Haarhaus, et al.Pageof 9