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Immunology and Cell Biology|August 15, 2018
Constitutive overexpression of TNF in BPSM1 mice causes iBALT and bone marrow nodular lymphocytic hyperplasiaCyril Seillet, Elyas H Arvell, Derek Lacey, et al.Annals of the New York Academy of Sciences|April 16, 2010
Antiapoptotic molecule Bcl-2 is essential for the anabolic activity of parathyroid hormone in boneYuichi Nagase, Mitsuyasu Iwasawa, Toru Akiyama, et al.Proceedings of the National Academy of Sciences of the United States of America|December 24, 2013
Enhanced stability of Mcl1, a prosurvival Bcl2 relative, blunts stress-induced apoptosis, causes male sterility, and promotes tumorigenesisToru Okamoto, Leigh Coultas, Donald Metcalf, et al.Journal of Immunology (Baltimore, Md. : 1950)|August 24, 2005
NKT cell stimulation with glycolipid antigen in vivo: costimulation-dependent expansion, Bim-dependent contraction, and hyporesponsiveness to further antigenic challengeAdam P Uldrich, Nadine Y Crowe, Konstantinos Kyparissoudis, et al.Gastroenterology|July 16, 2008
Intrahepatic murine CD8 T-cell activation associates with a distinct phenotype leading to Bim-dependent deathLauren E Holz, Volker Benseler, David G Bowen, et al.Molecular and Cellular Biology|June 2, 2006
The RUNX3 tumor suppressor upregulates Bim in gastric epithelial cells undergoing transforming growth factor beta-induced apoptosisTakashi Yano, Kosei Ito, Hiroshi Fukamachi, et al.Journal of Cell Science|May 31, 2007
Hrk/DP5 contributes to the apoptosis of select neuronal populations but is dispensable for haematopoietic cell apoptosisLeigh Coultas, Susanna Terzano, Tim Thomas, et al.Reproduction (Cambridge, England)|February 27, 2016
BCL2-modifying factor promotes germ cell loss during murine oogenesisKavitha Vaithiyanathan, Seng H Liew, Nadeen Zerafa, et al.The Journal of Cell Biology|August 5, 2009
The role of BH3-only protein Bim extends beyond inhibiting Bcl-2-like prosurvival proteinsDelphine Mérino, Maybelline Giam, Peter D Hughes, et al.The Journal of Biological Chemistry|October 23, 2009
Anti-apoptotic molecule Bcl-2 regulates the differentiation, activation, and survival of both osteoblasts and osteoclastsYuichi Nagase, Mitsuyasu Iwasawa, Toru Akiyama, et al.Pageof 10