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Diabetes|December 5, 2009
Glucose induces pancreatic islet cell apoptosis that requires the BH3-only proteins Bim and Puma and multi-BH domain protein BaxMark D McKenzie, Emma Jamieson, Elisa S Jansen, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 8, 2007
Two molecular pathways initiate mitochondria-dependent dopaminergic neurodegeneration in experimental Parkinson's diseaseCeline Perier, Jordi Bové, Du-Chu Wu, et al.
Nature|July 24, 2009
XIAP discriminates between type I and type II FAS-induced apoptosisPhilipp J Jost, Stephanie Grabow, Daniel Gray, et al.
Molecular Cell|September 25, 2012
DNA damage-induced primordial follicle oocyte apoptosis and loss of fertility require TAp63-mediated induction of Puma and NoxaJeffrey B Kerr, Karla J Hutt, Ewa M Michalak, et al.
Cell Death and Differentiation|August 12, 2021
Dual roles for LUBAC signaling in thymic epithelial cell development and survivalReema Jain, Kelin Zhao, Julie M Sheridan, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 26, 2006
Bim and Bad mediate imatinib-induced killing of Bcr/Abl+ leukemic cells, and resistance due to their loss is overcome by a BH3 mimeticJunya Kuroda, Hamsa Puthalakath, Mark S Cragg, et al.
Plos One|June 16, 2011
Type I interferon drives dendritic cell apoptosis via multiple BH3-only proteins following activation by PolyIC in vivoSilvia A Fuertes Marraco, Clare L Scott, Philippe Bouillet, et al.
Immunity|January 3, 2009
Fatal hepatitis mediated by tumor necrosis factor TNFalpha requires caspase-8 and involves the BH3-only proteins Bid and BimThomas Kaufmann, Philipp J Jost, Marc Pellegrini, et al.
Cell Reports|April 27, 2024
Mis-expression of GATA6 re-programs cell fate during early hematopoiesisCindy Audiger, Yacine Laâbi, Junli Nie, et al.
The Journal of Cell Biology|January 23, 2008
A novel BH3 ligand that selectively targets Mcl-1 reveals that apoptosis can proceed without Mcl-1 degradationErinna F Lee, Peter E Czabotar, Mark F van Delft, et al.
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