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Journal of Analytical Toxicology|June 29, 2006
A novel LC-ESI-MS-MS method for sensitive quantification of colchicine in human plasma: application to two case reportsEmuri Abe, Anne-Sophie Lemaire-Hurtel, Charlotte Duverneuil, et al.
Fertility and Sterility|June 9, 2015
Involvement of estrogen-related receptor-γ and mitochondrial content in intrauterine growth restriction and preeclampsiaDorothée Poidatz, Esther Dos Santos, Fabien Duval, et al.
Blood Coagulation & Fibrinolysis : an International Journal in Haemostasis and Thrombosis|April 16, 2003
Fibrinogen Poissy II (gammaN361K): a novel dysfibrinogenemia associated with defective polymerization and peptide B releaseFlorence Mathonnet, Laurent Guillon, Hélène Detruit, et al.
Reproductive Toxicology (Elmsford, N.Y.)|May 31, 2011
Antiproliferative and proapoptotic effects of bisphenol A on human trophoblastic JEG-3 cellsLucie Morice, Delphine Benaîtreau, Marie-Noëlle Dieudonné, et al.
American Journal of Human Genetics|September 12, 2007
Mutation in WNT10A is associated with an autosomal recessive ectodermal dysplasia: the odonto-onycho-dermal dysplasiaLynn Adaimy, Eliane Chouery, Hala Megarbane, et al.
Blood Coagulation & Fibrinolysis : an International Journal in Haemostasis and Thrombosis|April 7, 2007
Severe prekallikrein deficiencies due to homozygous C529Y mutationsDominique François, Nawel Trigui, Guillaume Leterreux, et al.
Biology of Reproduction|September 19, 2014
Preimplantation factor (PIF) promotes human trophoblast invasionHadia Moindjie, Esther Dos Santos, Laurence Loeuillet, et al.
Blood Coagulation & Fibrinolysis : an International Journal in Haemostasis and Thrombosis|August 29, 2009
Molecular characterization of a novel mutation in the factor XIII a subunit gene associated with a severe defect: importance of prophylactic substitutionPierre Morange, Nawel Trigui, Corinne Frère, et al.
The Journal of Endocrinology|August 3, 2010
Effects of adiponectin on human trophoblast invasionDelphine Benaitreau, Esther Dos Santos, Marie-Christine Leneveu, et al.
BMC Medical Genetics|June 9, 2009
Mild forms of hypophosphatasia mostly result from dominant negative effect of severe alleles or from compound heterozygosity for severe and moderate allelesDelphine Fauvert, Isabelle Brun-Heath, Anne-Sophie Lia-Baldini, et al.
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