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Current Genetic Medicine Reports|August 28, 2018
Using High-Throughput Animal or Cell-Based Models to Functionally Characterize GWAS SignalsPierre Dourlen, Julien Chapuis, Jean-Charles LambertActa Neuropathologica|April 15, 2019
The new genetic landscape of Alzheimer's disease: from amyloid cascade to genetically driven synaptic failure hypothesis?Pierre Dourlen, Devrim Kilinc, Nicolas Malmanche, et al.Trends in Neurosciences|April 23, 2025
BIN1 and Alzheimer's disease: the tau connectionPierre Dourlen, Devrim Kilinc, Isabelle Landrieu, et al.International Journal of Alzheimer'S Disease|June 16, 2012
Drosophila models of tauopathies: what have we learned?Marc Gistelinck, Jean-Charles Lambert, Patrick Callaerts, et al.Neuroscience Letters|May 17, 2006
Association study of the PIN1 gene with Alzheimer's diseaseJean-Charles Lambert, Faiza Bensemain, Julien Chapuis, et al.Neurobiology of Aging|July 13, 2007
Association study of the paraoxonase 1 gene with the risk of developing Alzheimer's diseaseJulien Chapuis, Maud Boscher, Faiza Bensemain, et al.Cell Reports|January 22, 2013
TDP-43 loss-of-function causes neuronal loss due to defective steroid receptor-mediated gene program switching in DrosophilaLies Vanden Broeck, Marina Naval-Sánchez, Yoshitsugu Adachi, et al.Brain Communications|March 15, 2021
Pyk2 overexpression in postsynaptic neurons blocks amyloid β<sub>1-42</sub>-induced synaptotoxicity in microfluidic co-culturesDevrim Kilinc, Anaïs-Camille Vreulx, Tiago Mendes, et al.Methods in Molecular Biology (Clifton, N.J.)|November 29, 2014
In vivo assessment of neuronal cell death in DrosophilaPierre DourlenMethods in Molecular Biology (Clifton, N.J.)|March 21, 2024
Identification of Tau Toxicity Modifiers in the Drosophila EyePierre DourlenPageof 20