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The Journal of Biological Chemistry|October 19, 2005
The human enhancer blocker CTC-binding factor interacts with the transcription factor KaisoPierre-Antoine Defossez, Kevin F Kelly, Guillaume J P Filion, et al.
Molecular Cell|September 3, 2005
DNA binding selectivity of MeCP2 due to a requirement for A/T sequences adjacent to methyl-CpGRobert J Klose, Shireen A Sarraf, Lars Schmiedeberg, et al.
The EMBO Journal|May 3, 2008
Zbtb4 represses transcription of P21CIP1 and controls the cellular response to p53 activationAxel Weber, Judith Marquardt, David Elzi, et al.
Development (Cambridge, England)|November 19, 2004
Kaiso is a genome-wide repressor of transcription that is essential for amphibian developmentAlexey Ruzov, Donncha S Dunican, Anna Prokhortchouk, et al.
Nucleic Acids Research|February 13, 2019
Genetic screens reveal mechanisms for the transcriptional regulation of tissue-specific genes in normal cells and tumorsIkrame Naciri, Marthe Laisné, Laure Ferry, et al.
Genome Research|November 2, 2023
E4F1 and ZNF148 are transcriptional activators of the -57A > C and wild-type TERT promoterBoon Haow Chua, Nurkaiyisah Zaal Anuar, Laure Ferry, et al.
Bioorganic & Medicinal Chemistry|November 21, 2021
Structure-based screening combined with computational and biochemical analyses identified the inhibitor targeting the binding of DNA Ligase 1 to UHRF1Satomi Kori, Yuki Shibahashi, Toru Ekimoto, et al.
Structure (London, England : 1993)|January 15, 2019
Structure of the UHRF1 Tandem Tudor Domain Bound to a Methylated Non-histone Protein, LIG1, Reveals Rules for Binding and RegulationSatomi Kori, Laure Ferry, Shohei Matano, et al.
Nucleic Acids Research|December 28, 2013
Immortality, but not oncogenic transformation, of primary human cells leads to epigenetic reprogramming of DNA methylation and gene expressionKatrina Gordon, Thomas Clouaire, Xun X Bao, et al.
Proteomics|March 18, 2014
MBD5 and MBD6 interact with the human PR-DUB complex through their methyl-CpG-binding domainH Irem Baymaz, Alexandra Fournier, Sophie Laget, et al.
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