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Leukemia Research|May 15, 2013
XIAP downregulation promotes caspase-dependent inhibition of proteasome activity in AML cellsBing Z Carter, Duncan H Mak, Zhiqiang Wang, et al.
Cancer Research|January 25, 2019
An ARC-Regulated IL1β/Cox-2/PGE2/β-Catenin/ARC Circuit Controls Leukemia-Microenvironment Interactions and Confers Drug Resistance in AMLBing Z Carter, Po Yee Mak, Xiangmeng Wang, et al.
Haematologica|December 23, 2020
Targeting MCL-1 dysregulates cell metabolism and leukemia-stroma interactions and resensitizes acute myeloid leukemia to BCL-2 inhibitionBing Z Carter, Po Yee Mak, Wenjing Tao, et al.
Haematologica|August 3, 2019
Combined inhibition of MDM2 and BCR-ABL1 tyrosine kinase targets chronic myeloid leukemia stem/progenitor cells in a murine modelBing Z Carter, Po Yee Mak, Hong Mu, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|July 13, 2026
The novel hypomethylating agent NTX-301 reprograms epigenetic and Hippo signaling pathways and exhibits pre-clinical activity in venetoclax-resistant and TP53-mutant AMLBing Z Carter, Po Yee Mak, Suresh Satpati, et al.
Oncotarget|October 3, 2015
Synergistic effects of p53 activation via MDM2 inhibition in combination with inhibition of Bcl-2 or Bcr-Abl in CD34+ proliferating and quiescent chronic myeloid leukemia blast crisis cellsBing Z Carter, Po Yee Mak, Duncan H Mak, et al.
Molecular Cancer Therapeutics|May 15, 2020
Combinatorial Inhibition of Focal Adhesion Kinase and BCL-2 Enhances Antileukemia Activity of Venetoclax in Acute Myeloid LeukemiaXiangmeng Wang, Po Yee Mak, Hong Mu, et al.
Oncotarget|March 10, 2016
Anti-apoptotic ARC protein confers chemoresistance by controlling leukemia-microenvironment interactions through a NFκB/IL1β signaling networkBing Z Carter, Po Yee Mak, Ye Chen, et al.
Science Translational Medicine|September 9, 2016
Combined targeting of BCL-2 and BCR-ABL tyrosine kinase eradicates chronic myeloid leukemia stem cellsBing Z Carter, Po Yee Mak, Hong Mu, et al.
Medrxiv : the Preprint Server for Health Sciences|April 29, 2025
Somatic TP53 Mutations Drive T and NK Cell Dysfunction in AML and Can be Rescued by Reactivating Wild Type p53Li Li, Muharrem Muftuoglu, Edward Ayoub, et al.
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