Showing results (11-20 of 46) with videos related to

Sort By:
Pageof 5
Oncotarget|July 14, 2016
Cetuximab response in CRC patient-derived xenografts seems predicted by an expression based RAS pathway signatureSheng Guo, Dawei Chen, Xuesong Huang, et al.
The Journal of Endocrinology|September 1, 2007
Isoginkgetin enhances adiponectin secretion from differentiated adiposarcoma cells via a novel pathway involving AMP-activated protein kinaseGuohong Liu, Mirta Grifman, James Macdonald, et al.
International Journal of Cancer|February 19, 2016
NSCLC harboring EGFR exon-20 insertions after the regulatory C-helix of kinase domain responds poorly to known EGFR inhibitorsMengmeng Yang, Xiaoxi Xu, Jie Cai, et al.
Biochemical and Biophysical Research Communications|March 7, 2006
Mineralocorticoid receptor is involved in the regulation of genes responsible for hepatic glucose productionGuohong Liu, Mirta Grifman, Benjamin Keily, et al.
Zhongguo Zhong Yao Za Zhi = Zhongguo Zhongyao Zazhi = China Journal of Chinese Materia Medica|August 20, 2017
[Effect of oridonin on apoptosis and intracellular reactive oxygen species level in triple-negative breast cancer MDA-MB-231 cells]Qi Qi, Pei Zhang, Qi-Xiang Li, et al.
Current Opinion in Pharmacology|October 7, 2021
Preclinical pharmacology modeling of chimeric antigen receptor T therapiesRajendra Kumari, Xuesong Ouyang, Jingjing Wang, et al.
Oncotarget|October 30, 2015
A set of defined oncogenic mutation alleles seems to better predict the response to cetuximab in CRC patient-derived xenograft than KRAS 12/13 mutationsDawei Chen, Xuesong Huang, Jie Cai, et al.
Cancer Research Communications|March 6, 2023
Tumor Purity in Preclinical Mouse Tumor ModelsWubin Qian, Xiaobo Chen, Yanghui Sheng, et al.
Gastroenterology Research and Practice|April 9, 2016
Pancreatic Stenting Reduces Post-ERCP Pancreatitis and Biliary Sepsis in High-Risk Patients: A Randomized, Controlled StudyHe-Kun Yin, Hai-En Wu, Qi-Xiang Li, et al.
Investigational New Drugs|April 25, 2006
MDDD, a 4,9-diazapyrenium derivative, is selectively toxic to glioma cells by inducing growth arrest at G0/G1 independently of p53De-Hua Yu, James Macdonald, Steve Josephs, et al.
Pageof 5