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Molecular Neurobiology|June 27, 2016
Overexpression of PLK3 Mediates the Degradation of Abnormal Prion Proteins Dependent on Chaperone-Mediated AutophagyHui Wang, Chan Tian, Jing Sun, et al.Molecular Neurobiology|April 30, 2013
Abortive cell cycle events in the brains of scrapie-infected hamsters with remarkable decreases of PLK3/Cdc25C and increases of PLK1/cyclin B1Hui Wang, Chan Tian, Yin Xu, et al.Scientific Reports|October 2, 2015
Activation of the AMPK-ULK1 pathway plays an important role in autophagy during prion infectionXue-Yu Fan, Chan Tian, Hui Wang, et al.Journal of Immunology (Baltimore, Md. : 1950)|May 21, 2005
Cutting edge: generation of splenic CD8+ and CD8- dendritic cell equivalents in Fms-like tyrosine kinase 3 ligand bone marrow culturesShalin H Naik, Anna I Proietto, Nicholas S Wilson, et al.Chinese Medical Journal|June 13, 2019
Regulatory factor X5 promotes hepatocellular carcinoma progression by transactivating tyrosine 3-monooxygenase/tryptophan 5-monooxygenase activation protein theta and suppressing apoptosisDong-Bo Chen, Yang-Jing Zhao, Xue-Yan Wang, et al.The International Journal of Biochemistry & Cell Biology|March 1, 2015
Polo-like kinase 3 (PLK3) mediates the clearance of the accumulated PrP mutants transiently expressed in cultured cells and pathogenic PrP(Sc) in prion infected cell line via protein interactionHui Wang, Chan Tian, Xue-Yu Fan, et al.Scientific Reports|September 5, 2020
RFX5 promotes the progression of hepatocellular carcinoma through transcriptional activation of KDM4ADong-Bo Chen, Xing-Wang Xie, Yang-Jing Zhao, et al.Cancer Cell International|May 6, 2020
Roles of eIF3m in the tumorigenesis of triple negative breast cancerWei Han, Cong Zhang, Chun-Tao Shi, et al.Pageof 2