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Journal of Molecular Biology|May 9, 2008
Alternative relay domains of Drosophila melanogaster myosin differentially affect ATPase activity, in vitro motility, myofibril structure and muscle functionWilliam A Kronert, Corey M Dambacher, Aileen F Knowles, et al.Biology|August 26, 2022
Myosin Transducer Inter-Strand Communication Is Critical for Normal ATPase Activity and Myofibril StructureWilliam A Kronert, Karen H Hsu, Aditi Madan, et al.Plos Genetics|December 25, 2013
Huntington's disease induced cardiac amyloidosis is reversed by modulating protein folding and oxidative stress pathways in the Drosophila heartGirish C Melkani, Adriana S Trujillo, Raul Ramos, et al.Rare Diseases (Austin, Tex.)|March 5, 2016
Drosophila as a potential model to ameliorate mutant Huntington-mediated cardiac amyloidosisAdriana S Trujillo, Raul Ramos, Rolf Bodmer, et al.Biophysical Journal|September 23, 2008
Alternative versions of the myosin relay domain differentially respond to load to influence Drosophila muscle kineticsChaoxing Yang, Seemanti Ramanath, William A Kronert, et al.Journal of Molecular Biology|November 1, 2011
Structural basis for myopathic defects engendered by alterations in the myosin rodAnthony Cammarato, Xiaochuan Edward Li, Mary C Reedy, et al.Molecular Biology of the Cell|November 1, 2018
Reductions in ATPase activity, actin sliding velocity, and myofibril stability yield muscle dysfunction in Drosophila models of myosin-based Freeman-Sheldon syndromeDeepti S Rao, William A Kronert, Yiming Guo, et al.FEBS Letters|October 1, 2017
TRiC/CCT chaperonins are essential for maintaining myofibril organization, cardiac physiological rhythm, and lifespanGirish C Melkani, Shruti Bhide, Andrew Han, et al.Journal of Cellular and Molecular Medicine|January 10, 2012
Measuring passive myocardial stiffness in Drosophila melanogaster to investigate diastolic dysfunctionGaurav Kaushik, Alexander C Zambon, Alexander Fuhrmann, et al.Proceedings of the National Academy of Sciences of the United States of America|July 16, 2005
Paramyosin phosphorylation site disruption affects indirect flight muscle stiffness and power generation in Drosophila melanogasterHongjun Liu, Mark S Miller, Douglas M Swank, et al.Pageof 181