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Clinical and Experimental Pharmacology & Physiology|September 3, 2015
Poly(ADP-ribose)polymerase 1 inhibition protects against age-dependent endothelial dysfunctionGuang-hao Zhang, Min Chao, Long-hua Hui, et al.
Digestive Diseases and Sciences|April 2, 2013
XRCC1 Arg399Gln, Arg194Trp, and Arg280His polymorphisms in esophageal cancer risk: a meta-analysisShan Li, Yan Deng, Jian-Peng You, et al.
International Journal of Cardiology|February 4, 2014
Inhibition of high-mobility group box 1 improves myocardial fibrosis and dysfunction in diabetic cardiomyopathyWen-Ke Wang, Ben Wang, Qing-Hua Lu, et al.
Oncotarget|April 23, 2016
Inhibition of myocyte-specific enhancer factor 2A improved diabetic cardiac fibrosis partially by regulating endothelial-to-mesenchymal transitionXue-Ying Chen, Rui-Juan Lv, Wei Zhang, et al.
Oncotarget|December 2, 2015
Prohibitin overexpression improves myocardial function in diabetic cardiomyopathyWen-qian Dong, Min Chao, Qing-hua Lu, et al.
Clinical Nephrology|July 16, 2013
Circulating levels of β2-microglobulin and cystatin C are associated with left atrial size: additional link between the kidney and the heartYu-Sheng Liu, Xin Wang, Wei-Dong Jiang, et al.
Yonsei Medical Journal|October 27, 2011
Association of lipoprotein-associated phospholipase A₂ with characteristics of vulnerable coronary atherosclerotic plaquesYu-Sheng Liu, Xiao-Bo Hu, Hong-Zhuan Li, et al.
Journal of Cellular and Molecular Medicine|September 12, 2014
HMGB1 mediates hyperglycaemia-induced cardiomyocyte apoptosis via ERK/Ets-1 signalling pathwayWen-Ke Wang, Qing-Hua Lu, Jia-Ning Zhang, et al.
Zhonghua Nei Ke Za Zhi|December 4, 2009
[The correlation of human serum Lp-PLA2 and hs-CRP and stability of coronary atherosclerotic plaques]Dong-Ling Xu, Jun-Ni Liu, Yi-Meng Du, et al.
Journal of Cellular and Molecular Medicine|October 25, 2017
Irisin inhibits high glucose-induced endothelial-to-mesenchymal transition and exerts a dose-dependent bidirectional effect on diabetic cardiomyopathyXue Liu, Haroon Mujahid, Bing Rong, et al.
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