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Scientific Reports|November 14, 2015
TAK1 Regulates Myocardial Response to Pathological Stress via NFAT, NFκB, and Bnip3 PathwaysLei Li, Yi Chen, Jing Li, et al.
Journal of Molecular and Cellular Cardiology|June 20, 2020
CYLD exaggerates pressure overload-induced cardiomyopathy via suppressing autolysosome efflux in cardiomyocytesLei Qi, Huimei Zang, Weiwei Wu, et al.
Biorxiv : the Preprint Server for Biology|November 24, 2025
Lipid peroxidation intrinsically induces mitochondrial iron overload via Bach1-HO-1 signaling to promote cardiac ferroptosisXiaoyun Guo, Yi Chen, Yachang Zeng, et al.
Scientific Reports|September 19, 2023
Oxidative stress induces mitochondrial iron overload and ferroptotic cell deathYi Chen, Xiaoyun Guo, Yachang Zeng, et al.
Free Radical Biology & Medicine|July 27, 2023
Ferroptosis contributes to catecholamine-induced cardiotoxicity and pathological remodelingYi Chen, Xiaoyun Guo, Yachang Zeng, et al.
The Journal of Clinical Investigation|January 6, 2022
TAB2 deficiency induces dilated cardiomyopathy by promoting RIPK1-dependent apoptosis and necroptosisHaifeng Yin, Xiaoyun Guo, Yi Chen, et al.
The Journal of Biological Chemistry|August 11, 2024
Anchored PKA synchronizes adrenergic phosphoregulation of cardiac Cav1.2 channelsLipeng Wang, Yi Chen, Jin Li, et al.
Free Radical Biology & Medicine|August 4, 2020
NFκB promotes oxidative stress-induced necrosis and ischemia/reperfusion injury by inhibiting Nrf2-ARE pathwayXiaoyun Guo, Siqi Hong, Hui He, et al.
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