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Qingjian He

Showing results (1-10 of 10) with videos related to

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Brazilian Journal of Otorhinolaryngology|March 1, 2025
Clinical study of the diagnosis of thyroid tumours using Raman spectroscopyQingjian He, Lianjin Qin, Yongqiang Yao, et al.
Translational Cancer Research|September 15, 2025
Investigation into the sensitivity of adipocytes mediated by the major vault protein (MVP) to chemotherapy for triple-negative breast cancerLianjin Qin, Ruofan Fei, Wenjuan Wang, et al.
Micromachines|May 30, 2019
The Matrix KV Storage System Based on NVM DevicesTao Cai, Fuli Chen, Qingjian He, et al.
Micromachines|December 5, 2020
A New Embedded Key-Value Store for NVM Device SimulatorTao Cai, Qingjian He, Dejiao Niu, et al.
Frontiers in Pharmacology|February 26, 2024
Microvascular rarefaction caused by the NOTCH signaling pathway is a key cause of TKI-apatinib-induced hypertension and cardiac damageWenJuan Wang, Guodong Li, Jie Ma, et al.
Journal of Molecular Histology|June 29, 2026
Alpha-2-macroglobulin attenuates glucocorticoid-induced osteonecrosis of the femoral head by enhancing osteogenesis and antioxidant defense via the Akt/Nrf2 signaling pathwayQingjian He, Xin Zheng, Boyun Huang, et al.
Annals of Translational Medicine|June 24, 2021
A narrative review on the interaction between genes and the treatment of hypertension and breast cancerWenjuan Wang, Qingjian He, Haodong Zhang, et al.
Applied Biochemistry and Biotechnology|July 1, 2022
Apatinib Through Activating the RhoA/ROCK Signaling Pathway to Cause Dysfunction of Vascular Smooth Muscle CellsWenjuan Wang, Qingjian He, Chenchen Zhuang, et al.
Frontiers in Cardiovascular Medicine|July 11, 2022
Research on the Mechanism and Prevention of Hypertension Caused by Apatinib Through the RhoA/ROCK Signaling Pathway in a Mouse Model of Gastric CancerWenjuan Wang, Qingjian He, Caie Li, et al.
Frontiers in Pharmacology|January 24, 2025
Endothelin receptor antagonists (ERAs) can potentially be used as therapeutic drugs to reduce hypertension caused by small molecule tyrosine kinase inhibitors (TKIs)Qingjian He, Junling Lin, Chanjuan Mo, et al.
Pageof 1

Showing results (1-10 of 10) with videos related to

Sort By:
Pageof 1
Brazilian Journal of Otorhinolaryngology|March 1, 2025
Clinical study of the diagnosis of thyroid tumours using Raman spectroscopyQingjian He, Lianjin Qin, Yongqiang Yao, et al.
Translational Cancer Research|September 15, 2025
Investigation into the sensitivity of adipocytes mediated by the major vault protein (MVP) to chemotherapy for triple-negative breast cancerLianjin Qin, Ruofan Fei, Wenjuan Wang, et al.
Micromachines|May 30, 2019
The Matrix KV Storage System Based on NVM DevicesTao Cai, Fuli Chen, Qingjian He, et al.
Micromachines|December 5, 2020
A New Embedded Key-Value Store for NVM Device SimulatorTao Cai, Qingjian He, Dejiao Niu, et al.
Frontiers in Pharmacology|February 26, 2024
Microvascular rarefaction caused by the NOTCH signaling pathway is a key cause of TKI-apatinib-induced hypertension and cardiac damageWenJuan Wang, Guodong Li, Jie Ma, et al.
Journal of Molecular Histology|June 29, 2026
Alpha-2-macroglobulin attenuates glucocorticoid-induced osteonecrosis of the femoral head by enhancing osteogenesis and antioxidant defense via the Akt/Nrf2 signaling pathwayQingjian He, Xin Zheng, Boyun Huang, et al.
Annals of Translational Medicine|June 24, 2021
A narrative review on the interaction between genes and the treatment of hypertension and breast cancerWenjuan Wang, Qingjian He, Haodong Zhang, et al.
Applied Biochemistry and Biotechnology|July 1, 2022
Apatinib Through Activating the RhoA/ROCK Signaling Pathway to Cause Dysfunction of Vascular Smooth Muscle CellsWenjuan Wang, Qingjian He, Chenchen Zhuang, et al.
Frontiers in Cardiovascular Medicine|July 11, 2022
Research on the Mechanism and Prevention of Hypertension Caused by Apatinib Through the RhoA/ROCK Signaling Pathway in a Mouse Model of Gastric CancerWenjuan Wang, Qingjian He, Caie Li, et al.
Frontiers in Pharmacology|January 24, 2025
Endothelin receptor antagonists (ERAs) can potentially be used as therapeutic drugs to reduce hypertension caused by small molecule tyrosine kinase inhibitors (TKIs)Qingjian He, Junling Lin, Chanjuan Mo, et al.
Pageof 1