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ACS Nano|October 20, 2025
Piezo-Nanowired Stem Cells: Ultrasound-Powered Neuronal Commitment for Rapid Neural Circuit Reconstruction after Traumatic Brain InjuryKeyi Li, Wenhan Wang, Wenjun Ma, et al.
International Immunopharmacology|May 8, 2025
GRK2-facilitated TLR4 signaling promotes cardiac fibrosis in rheumatic miceZhen Wang, Rui Chu, Hui Ge, et al.
CNS Neuroscience & Therapeutics|October 10, 2024
Ultra-early stage lower-grade gliomas: How can we define and differentiate these easily misdiagnosed gliomas through intraoperative molecular diagnosisZhe Han, Qingtong Wang, Jia Li, et al.
Cancer Letters|May 7, 2026
Levetiracetam Reverses Temozolomide Resistance in Glioblastoma by Blocking Drug Efflux through RAB5A/CD63-RAB35 AxisJixing Zhao, Mingkun Li, Qinran Zhang, et al.
Scientific Reports|March 29, 2017
Paroxetine alleviates T lymphocyte activation and infiltration to joints of collagen-induced arthritisQingtong Wang, Longsheng Wang, Li Wu, et al.
European Journal of Pharmacology|March 5, 2013
Comparative efficacy of TACI-Ig with TNF-alpha inhibitor and methotrexate in DBA/1 mice with collagen-induced arthritisLingling Zhang, Peipei Li, Shanshan Song, et al.
Journal of Ethnopharmacology|May 9, 2022
Ziyuglycoside I attenuates collagen-induced arthritis through inhibiting plasma cell expansionHanfei Sun, Manman Wang, Tiantian Su, et al.
Inflammation Research : Official Journal of the European Histamine Research Society ... [Et Al.]|November 4, 2022
Attenuation of experimental osteoarthritis with human adipose-derived mesenchymal stem cell therapy: inhibition of the pyroptosis in chondrocytesLiang Xu, Feng Zhang, Gang Cheng, et al.
Pharmaceuticals (Basel, Switzerland)|May 27, 2023
GRK2 Mediates Macrophage Polarization by Regulating EP4-cAMP-pCREB Signaling in Ulcerative Colitis and the Therapeutic Effect of Paroxetine on Mice with DSS-Induced ColitisJiawei Zhang, Xianzheng Zhang, Mingdian Lu, et al.
Cell Communication and Signaling : CCS|December 1, 2023
CP-25 inhibits the hyperactivation of rheumatic synoviocytes by suppressing the switch in G<sub>αs</sub>-G<sub>αi</sub> coupling to the β<sub>2</sub>-adrenergic receptorMingli Ge, Li Wu, Feng He, et al.
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