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Journal of Virology|January 26, 2018
Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBVQuentin Bazot, Kostas Paschos, Martin J Allday
Current Topics in Microbiology and Immunology|October 3, 2015
The EBNA3 Family: Two Oncoproteins and a Tumour Suppressor that Are Central to the Biology of EBV in B CellsMartin J Allday, Quentin Bazot, Robert E White
Nucleic Acids Research|January 17, 2019
Requirement for PRC1 subunit BMI1 in host gene activation by Epstein-Barr virus protein EBNA3CKostas Paschos, Quentin Bazot, Jonathan Lees, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|April 9, 2019
Phosphodiesterase-induced cAMP degradation restricts hepatitis B virus infectionAntonia Alexandra Evripioti, Ana Maria Ortega-Prieto, Jessica Katy Skelton, et al.
Nucleic Acids Research|December 2, 2016
Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expressionKostas Paschos, Quentin Bazot, Guiyi Ho, et al.
Plos Pathogens|January 12, 2016
EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B CellsJens S Kalchschmidt, Adam C T Gillman, Kostas Paschos, et al.
Plos Biology|August 4, 2017
EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latencyChristine T Styles, Quentin Bazot, Gillian A Parker, et al.
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