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Proceedings of the National Academy of Sciences of the United States of America
|
May 1, 1991
Reversal of age-related increase in brain protein oxidation, decrease in enzyme activity, and loss in temporal and spatial memory by chronic administration of the spin-trapping compound N-tert-butyl-alpha-phenylnitrone
J M Carney, P E Starke-Reed, C N Oliver, et al.
Brain Research
|
January 22, 2000
Protection of methamphetamine nigrostriatal toxicity by dietary selenium
H C Kim, W K Jhoo, D Y Choi, et al.
American Journal of Otolaryngology
|
May 1, 1985
Factors influencing collagen-induced autoimmune ear disease
T J Yoo, R A Floyd, N Sudo, et al.
Free Radical Biology & Medicine
|
January 1, 1997
Proteins but not nucleic acids are molecular targets for the free radical attack during reoxygenation of rat hepatocytes
P Caraceni, N De Maria, H S Ryu, et al.
Carcinogenesis
|
April 28, 2000
Dietary choline restriction causes complex I dysfunction and increased H(2)O(2) generation in liver mitochondria
K Hensley, Y Kotake, H Sang, et al.
Cancer Research
|
October 27, 1998
Inhibition by phenyl N-tert-butyl nitrone of early phase carcinogenesis in the livers of rats fed a choline-deficient, L-amino acid-defined diet
D Nakae, Y Kotake, H Kishida, et al.
The Journals of Gerontology. Series A, Biological Sciences and Medical Sciences
|
June 29, 2002
Caloric restriction mimetics: metabolic interventions
R Weindruch, K P Keenan, J M Carney, et al.
Journal of Neurochemistry
|
April 27, 1999
p38 kinase is activated in the Alzheimer's disease brain
K Hensley, R A Floyd, N Y Zheng, et al.
Digestive Diseases and Sciences
|
October 1, 1996
Increased oxidative stress and decreased antioxidant defenses in mucosa of inflammatory bowel disease
L Lih-Brody, S R Powell, K P Collier, et al.
Free Radical Biology & Medicine
|
February 22, 2005
Biomarkers of oxidative stress study II: are oxidation products of lipids, proteins, and DNA markers of CCl4 poisoning?
M B Kadiiska, B C Gladen, D D Baird, et al.
Page
of 18
Search research articles
Search
Showing results (171-180 of 180) with videos related to
Sort By:
Page
of 18
You have reached the last page of results.
This site can display upto 180 results.
Proceedings of the National Academy of Sciences of the United States of America
|
May 1, 1991
Reversal of age-related increase in brain protein oxidation, decrease in enzyme activity, and loss in temporal and spatial memory by chronic administration of the spin-trapping compound N-tert-butyl-alpha-phenylnitrone
J M Carney, P E Starke-Reed, C N Oliver, et al.
Brain Research
|
January 22, 2000
Protection of methamphetamine nigrostriatal toxicity by dietary selenium
H C Kim, W K Jhoo, D Y Choi, et al.
American Journal of Otolaryngology
|
May 1, 1985
Factors influencing collagen-induced autoimmune ear disease
T J Yoo, R A Floyd, N Sudo, et al.
Free Radical Biology & Medicine
|
January 1, 1997
Proteins but not nucleic acids are molecular targets for the free radical attack during reoxygenation of rat hepatocytes
P Caraceni, N De Maria, H S Ryu, et al.
Carcinogenesis
|
April 28, 2000
Dietary choline restriction causes complex I dysfunction and increased H(2)O(2) generation in liver mitochondria
K Hensley, Y Kotake, H Sang, et al.
Cancer Research
|
October 27, 1998
Inhibition by phenyl N-tert-butyl nitrone of early phase carcinogenesis in the livers of rats fed a choline-deficient, L-amino acid-defined diet
D Nakae, Y Kotake, H Kishida, et al.
The Journals of Gerontology. Series A, Biological Sciences and Medical Sciences
|
June 29, 2002
Caloric restriction mimetics: metabolic interventions
R Weindruch, K P Keenan, J M Carney, et al.
Journal of Neurochemistry
|
April 27, 1999
p38 kinase is activated in the Alzheimer's disease brain
K Hensley, R A Floyd, N Y Zheng, et al.
Digestive Diseases and Sciences
|
October 1, 1996
Increased oxidative stress and decreased antioxidant defenses in mucosa of inflammatory bowel disease
L Lih-Brody, S R Powell, K P Collier, et al.
Free Radical Biology & Medicine
|
February 22, 2005
Biomarkers of oxidative stress study II: are oxidation products of lipids, proteins, and DNA markers of CCl4 poisoning?
M B Kadiiska, B C Gladen, D D Baird, et al.
Page
of 18