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Metabolic Brain Disease|March 1, 1986
Brain monoamines after portacaval anastomosisA M Mans, R A HawkinsThe American Journal of Physiology|November 1, 1991
Regional blood-brain barrier transport of ketone bodies in portacaval-shunted ratsR A Hawkins, A M MansThe American Journal of Physiology|February 1, 1986
Regional ketone body utilization by rat brain in starvation and diabetesR A Hawkins, A M Mans, D W DavisJournal of Neurochemistry|November 1, 1994
Metabolic abnormalities and grade of encephalopathy in acute hepatic failureA M Mans, M R DeJoseph, R A HawkinsMetabolic Brain Disease|December 1, 1987
Regional brain glucose use in unstressed rats after two days of starvationA M Mans, D W Davis, R A HawkinsThe American Journal of Physiology|January 1, 1982
Amino acid supply to individual cerebral structures in awake and anesthetized ratsR A Hawkins, A M Mans, J F BiebuyckMetabolic Brain Disease|June 1, 1986
Regional blood-brain barrier transport of glucose after portacaval anastomosisA M Mans, D W Davis, R A HawkinsThe American Journal of Physiology|July 1, 1983
Ammonia selectively stimulates neutral amino acid transport across blood-brain barrierA M Mans, J F Biebuyck, R A HawkinsJournal of Neurochemistry|March 1, 1982
Regional blood-brain barrier permeability to amino acids after portacaval anastomosisA M Mans, J F Biebuyck, K Shelly, et al.The Biochemical Journal|December 1, 1990
Hyperammonaemia causes many of the changes found after portacaval shuntingJ Jessy, A M Mans, M R DeJoseph, et al.Pageof 24