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R A Khalil

Showing results (11-20 of 42) with videos related to

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The Journal of Pharmacology and Experimental Therapeutics|September 22, 1999
Decreased [Ca(2+)](i) during inhibition of coronary smooth muscle contraction by 17beta-estradiol, progesterone, and testosteroneJ G Murphy, R A Khalil
Clinical and Experimental Pharmacology & Physiology|September 28, 1999
Gender-specific inhibition of Ca2+ entry mechanisms of arterial vasoconstriction by sex hormonesJ K Crews, R A Khalil
Phlebology|May 6, 2008
Mechanisms of varicose vein formation: valve dysfunction and wall dilationJ D Raffetto, R A Khalil
Circulation Research|December 1, 1991
Imaging of protein kinase C distribution and translocation in living vascular smooth muscle cellsR A Khalil, K G Morgan
Advances in Pharmacology (San Diego, Calif.)|February 19, 2017
Protein Kinase C as Regulator of Vascular Smooth Muscle Function and Potential Target in Vascular DisordersH C Ringvold, R A Khalil
Advances in Pharmacology (San Diego, Calif.)|July 25, 2016
Mechanisms of Endothelial Dysfunction in Hypertensive Pregnancy and PreeclampsiaJ S Possomato-Vieira, R A Khalil
Hypertension (Dallas, Tex. : 1979)|June 1, 1989
Agonist-induced vascular toneJ Nishimura, R A Khalil, C van Breemen
The American Journal of Physiology|June 1, 1994
In situ determination of [Ca2+]i threshold for translocation of the alpha-protein kinase C isoformR A Khalil, C Lajoie, K G Morgan
Hypertension (Dallas, Tex. : 1979)|March 7, 2001
Endothelin-1 enhances eicosanoids-induced coronary smooth muscle contraction by activating specific protein kinase C isoformsZ N Sirous, J B Fleming, R A Khalil
Journal of Cardiovascular Pharmacology|March 10, 2000
Preconditioning of coronary artery against vasoconstriction by endothelin-1 and prostaglandin F2alpha during repeated downregulation of epsilon-protein kinase CC A Kanashiro, K A Altirkawi, R A Khalil
Pageof 5

Showing results (11-20 of 42) with videos related to

Sort By:
Pageof 5
The Journal of Pharmacology and Experimental Therapeutics|September 22, 1999
Decreased [Ca(2+)](i) during inhibition of coronary smooth muscle contraction by 17beta-estradiol, progesterone, and testosteroneJ G Murphy, R A Khalil
Clinical and Experimental Pharmacology & Physiology|September 28, 1999
Gender-specific inhibition of Ca2+ entry mechanisms of arterial vasoconstriction by sex hormonesJ K Crews, R A Khalil
Phlebology|May 6, 2008
Mechanisms of varicose vein formation: valve dysfunction and wall dilationJ D Raffetto, R A Khalil
Circulation Research|December 1, 1991
Imaging of protein kinase C distribution and translocation in living vascular smooth muscle cellsR A Khalil, K G Morgan
Advances in Pharmacology (San Diego, Calif.)|February 19, 2017
Protein Kinase C as Regulator of Vascular Smooth Muscle Function and Potential Target in Vascular DisordersH C Ringvold, R A Khalil
Advances in Pharmacology (San Diego, Calif.)|July 25, 2016
Mechanisms of Endothelial Dysfunction in Hypertensive Pregnancy and PreeclampsiaJ S Possomato-Vieira, R A Khalil
Hypertension (Dallas, Tex. : 1979)|June 1, 1989
Agonist-induced vascular toneJ Nishimura, R A Khalil, C van Breemen
The American Journal of Physiology|June 1, 1994
In situ determination of [Ca2+]i threshold for translocation of the alpha-protein kinase C isoformR A Khalil, C Lajoie, K G Morgan
Hypertension (Dallas, Tex. : 1979)|March 7, 2001
Endothelin-1 enhances eicosanoids-induced coronary smooth muscle contraction by activating specific protein kinase C isoformsZ N Sirous, J B Fleming, R A Khalil
Journal of Cardiovascular Pharmacology|March 10, 2000
Preconditioning of coronary artery against vasoconstriction by endothelin-1 and prostaglandin F2alpha during repeated downregulation of epsilon-protein kinase CC A Kanashiro, K A Altirkawi, R A Khalil
Pageof 5