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R A Mackenzie

Showing results (121-130 of 155) with videos related to

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Journal of Neural Transmission (Vienna, Austria : 1996)|December 17, 2009
Neuropathological heterogeneity in frontotemporal lobar degeneration with TDP-43 proteinopathy: a quantitative study of 94 cases using principal components analysisRichard A Armstrong, William Ellis, Ronald L Hamilton, et al.
Acta Neuropathologica|June 7, 2011
A harmonized classification system for FTLD-TDP pathologyIan R A Mackenzie, Manuela Neumann, Atik Baborie, et al.
Neuropathology : Official Journal of the Japanese Society of Neuropathology|September 10, 2015
Two cases of rheumatoid meningitisShino Magaki, Edward Chang, Robert R Hammond, et al.
Neurobiology of Disease|November 9, 2011
Synaptic dysfunction in progranulin-deficient miceTerri L Petkau, Scott J Neal, Austen Milnerwood, et al.
Acta Neuropathologica|February 6, 2013
hnRNP A3 binds to GGGGCC repeats and is a constituent of p62-positive/TDP43-negative inclusions in the hippocampus of patients with C9orf72 mutationsKohji Mori, Sven Lammich, Ian R A Mackenzie, et al.
Acta Neuropathologica Communications|August 5, 2018
Novel antibodies reveal presynaptic localization of C9orf72 protein and reduced protein levels in C9orf72 mutation carriersPetra Frick, Chantal Sellier, Ian R A Mackenzie, et al.
Brain : a Journal of Neurology|February 21, 2012
Clinical and pathological features of familial frontotemporal dementia caused by C9ORF72 mutation on chromosome 9pGing-Yuek R Hsiung, Mariely DeJesus-Hernandez, Howard H Feldman, et al.
The American Journal of Pathology|May 27, 2004
alpha-internexin is present in the pathological inclusions of neuronal intermediate filament inclusion diseaseNigel J Cairns, Victoria Zhukareva, Kunihiro Uryu, et al.
Acta Neuropathologica|June 2, 2004
alpha-Internexin aggregates are abundant in neuronal intermediate filament inclusion disease (NIFID) but rare in other neurodegenerative diseasesNigel J Cairns, Kunihiro Uryu, Eileen H Bigio, et al.
Brain : a Journal of Neurology|August 23, 2011
FET proteins TAF15 and EWS are selective markers that distinguish FTLD with FUS pathology from amyotrophic lateral sclerosis with FUS mutationsManuela Neumann, Eva Bentmann, Dorothee Dormann, et al.
Pageof 16

Showing results (121-130 of 155) with videos related to

Sort By:
Pageof 16
Journal of Neural Transmission (Vienna, Austria : 1996)|December 17, 2009
Neuropathological heterogeneity in frontotemporal lobar degeneration with TDP-43 proteinopathy: a quantitative study of 94 cases using principal components analysisRichard A Armstrong, William Ellis, Ronald L Hamilton, et al.
Acta Neuropathologica|June 7, 2011
A harmonized classification system for FTLD-TDP pathologyIan R A Mackenzie, Manuela Neumann, Atik Baborie, et al.
Neuropathology : Official Journal of the Japanese Society of Neuropathology|September 10, 2015
Two cases of rheumatoid meningitisShino Magaki, Edward Chang, Robert R Hammond, et al.
Neurobiology of Disease|November 9, 2011
Synaptic dysfunction in progranulin-deficient miceTerri L Petkau, Scott J Neal, Austen Milnerwood, et al.
Acta Neuropathologica|February 6, 2013
hnRNP A3 binds to GGGGCC repeats and is a constituent of p62-positive/TDP43-negative inclusions in the hippocampus of patients with C9orf72 mutationsKohji Mori, Sven Lammich, Ian R A Mackenzie, et al.
Acta Neuropathologica Communications|August 5, 2018
Novel antibodies reveal presynaptic localization of C9orf72 protein and reduced protein levels in C9orf72 mutation carriersPetra Frick, Chantal Sellier, Ian R A Mackenzie, et al.
Brain : a Journal of Neurology|February 21, 2012
Clinical and pathological features of familial frontotemporal dementia caused by C9ORF72 mutation on chromosome 9pGing-Yuek R Hsiung, Mariely DeJesus-Hernandez, Howard H Feldman, et al.
The American Journal of Pathology|May 27, 2004
alpha-internexin is present in the pathological inclusions of neuronal intermediate filament inclusion diseaseNigel J Cairns, Victoria Zhukareva, Kunihiro Uryu, et al.
Acta Neuropathologica|June 2, 2004
alpha-Internexin aggregates are abundant in neuronal intermediate filament inclusion disease (NIFID) but rare in other neurodegenerative diseasesNigel J Cairns, Kunihiro Uryu, Eileen H Bigio, et al.
Brain : a Journal of Neurology|August 23, 2011
FET proteins TAF15 and EWS are selective markers that distinguish FTLD with FUS pathology from amyotrophic lateral sclerosis with FUS mutationsManuela Neumann, Eva Bentmann, Dorothee Dormann, et al.
Pageof 16