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Fundamental and Applied Toxicology : Official Journal of the Society of Toxicology
|
August 1, 1993
Hepatocyte spheroids: prolonged hepatocyte viability for in vitro modeling of nongenotoxic carcinogenesis
R A Roberts, A R Soames
Journal of Cell Science
|
November 1, 1989
Bradykinin receptor number and sensitivity to ligand stimulation of mitogenesis is increased by expression of a mutant ras oncogene
R A Roberts, W J Gullick
Journal of Cell Science
|
November 27, 1999
Induction of DNA replication by peroxisome proliferators is independent of both tumour necrosis factor (alpha) priming and EGF-receptor tyrosine kinase activity
S Chevalier, N Macdonald, R A Roberts
Current Protein & Peptide Science
|
October 9, 2002
PPAR alpha-mediated responses in the rodent liver: a holistic biochemical view
S Chevalier, N Macdonald, R A Roberts
Annals of the Rheumatic Diseases
|
May 4, 1999
Rheumatic disease and the Australian aborigine
R A Roberts-Thomson, P J Roberts-Thomson
Cancer Research
|
October 16, 1999
Addition of peroxisome proliferator-activated receptor alpha to guinea pig hepatocytes confers increased responsiveness to peroxisome proliferators
N Macdonald, P R Holden, R A Roberts
IEEE Transactions on Ultrasonics, Ferroelectrics, and Frequency Control
|
January 1, 1989
Reciprocal relations for transmission coefficients: theory and application
J Qu, J D Achenbach, R A Roberts
Cancer Letters
|
June 29, 1995
Enhanced hepatocyte colony growth in soft agar after in vivo treatment with a genotoxic carcinogen: a potential assay for hepatocarcinogens?
N H James, J Ashby, R A Roberts
The Journal of Cell Biology
|
April 1, 1994
Suppression of liver cell apoptosis in vitro by the non-genotoxic hepatocarcinogen and peroxisome proliferator nafenopin
A C Bayly, R A Roberts, C Dive
Carcinogenesis
|
December 12, 1997
Tumour necrosis factor alpha (TNF alpha) suppresses apoptosis and induces DNA synthesis in rodent hepatocytes: a mediator of the hepatocarcinogenicity of peroxisome proliferators?
M Rolfe, N H James, R A Roberts
Page
of 11
Search research articles
Search
Showing results (31-40 of 110) with videos related to
Sort By:
Page
of 11
Fundamental and Applied Toxicology : Official Journal of the Society of Toxicology
|
August 1, 1993
Hepatocyte spheroids: prolonged hepatocyte viability for in vitro modeling of nongenotoxic carcinogenesis
R A Roberts, A R Soames
Journal of Cell Science
|
November 1, 1989
Bradykinin receptor number and sensitivity to ligand stimulation of mitogenesis is increased by expression of a mutant ras oncogene
R A Roberts, W J Gullick
Journal of Cell Science
|
November 27, 1999
Induction of DNA replication by peroxisome proliferators is independent of both tumour necrosis factor (alpha) priming and EGF-receptor tyrosine kinase activity
S Chevalier, N Macdonald, R A Roberts
Current Protein & Peptide Science
|
October 9, 2002
PPAR alpha-mediated responses in the rodent liver: a holistic biochemical view
S Chevalier, N Macdonald, R A Roberts
Annals of the Rheumatic Diseases
|
May 4, 1999
Rheumatic disease and the Australian aborigine
R A Roberts-Thomson, P J Roberts-Thomson
Cancer Research
|
October 16, 1999
Addition of peroxisome proliferator-activated receptor alpha to guinea pig hepatocytes confers increased responsiveness to peroxisome proliferators
N Macdonald, P R Holden, R A Roberts
IEEE Transactions on Ultrasonics, Ferroelectrics, and Frequency Control
|
January 1, 1989
Reciprocal relations for transmission coefficients: theory and application
J Qu, J D Achenbach, R A Roberts
Cancer Letters
|
June 29, 1995
Enhanced hepatocyte colony growth in soft agar after in vivo treatment with a genotoxic carcinogen: a potential assay for hepatocarcinogens?
N H James, J Ashby, R A Roberts
The Journal of Cell Biology
|
April 1, 1994
Suppression of liver cell apoptosis in vitro by the non-genotoxic hepatocarcinogen and peroxisome proliferator nafenopin
A C Bayly, R A Roberts, C Dive
Carcinogenesis
|
December 12, 1997
Tumour necrosis factor alpha (TNF alpha) suppresses apoptosis and induces DNA synthesis in rodent hepatocytes: a mediator of the hepatocarcinogenicity of peroxisome proliferators?
M Rolfe, N H James, R A Roberts
Page
of 11