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The Journal of Biological Chemistry|April 7, 2000
Bcl-2 inhibits a Fas-induced conformational change in the Bax N terminus and Bax mitochondrial translocationK M Murphy, U N Streips, R B Lock
Cancer Chemotherapy and Pharmacology|January 1, 1997
Potentiation of etoposide-induced apoptosis by staurosporine in human tumor cells is associated with events downstream of DNA-protein complex formationR B Lock, B S Thompson, D M Sullivan, et al.
International Journal of Radiation Biology and Related Studies in Physics, Chemistry, and Medicine|April 1, 1987
Effects of fractionated X-irradiation on subsequent response to acute X-irradiation in two human tumour cell lines in vitroA S Bellamy, R D Whelan, R B Lock, et al.
Cancer Research|September 15, 1994
Concentration-dependent differences in the mechanisms by which caffeine potentiates etoposide cytotoxicity in HeLa cellsR B Lock, O V Galperina, R C Feldhoff, et al.
Molecular and Cellular Biology|August 1, 1989
Depletion of topoisomerase II in isolated nuclei during a glucose-regulated stress responseJ W Shen, J R Subjeck, R B Lock, et al.
Cancer Research|December 15, 1991
Attenuated topoisomerase II content directly correlates with a low level of drug resistance in a Chinese hamster ovary cell lineC D Webb, M D Latham, R B Lock, et al.
Cell Death and Differentiation|March 14, 2000
Bcl-2 inhibits Bax translocation from cytosol to mitochondria during drug-induced apoptosis of human tumor cellsK M Murphy, V Ranganathan, M L Farnsworth, et al.
Leukemia|July 16, 2016
A review of new agents evaluated against pediatric acute lymphoblastic leukemia by the Pediatric Preclinical Testing ProgramL Jones, H Carol, K Evans, et al.
NCI Monographs : a Publication of the National Cancer Institute|January 1, 1988
Interactions between antitumor drugs and radiation in mammalian tumor cell lines: differential drug responses and mechanisms of resistance following fractionated X-irradiation or continuous drug exposure in vitroB T Hill, R D Whelan, L K Hosking, et al.
Leukemia|June 22, 2001
High level resistance to glucocorticoids, associated with a dysfunctional glucocorticoid receptor, in childhood acute lymphoblastic leukemia cells selected for methotrexate resistanceV S Catts, M L Farnsworth, M Haber, et al.
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