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R B Luftig

Showing results (81-90 of 106) with videos related to

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Virology|January 15, 1980
Preparations enriched for "immature" murine leukemia virus particles that remain in tissue culture fluids are deficient in Pr65gag proteolyic activityY Yoshinaka, K Ishigame, T Ohno, et al.
Nucleic Acids Research|November 25, 1989
Sequence of human enteric adenovirus type 41 Tak fiber protein geneN J Pieniazek, S B Slemenda, D Pieniazek, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 1, 1986
Myristylation site in Pr65gag is essential for virus particle formation by Moloney murine leukemia virusA Rein, M R McClure, N R Rice, et al.
Virology|August 1, 1980
Quantitation of the interaction between adenovirus types 2 and 5 and microtubules inside infected cellsB D Miles, R B Luftig, J A Weatherbee, et al.
Virology|January 1, 1990
Differential growth of human enteric adenovirus 41 (TAK) in continuous cell linesD Pieniazek, N J Pieniazek, D Macejak, et al.
International Immunology|November 1, 1996
Induction of apoptosis by protease-defective particle preparations of human immunodeficiency virus type 1 is specific to a subset of U937-derived subclonesM Kameoka, T Kimura, Q Zhong, et al.
Journal of Acquired Immune Deficiency Syndromes|June 1, 1993
A dominant epitope of HIV-1 protease recognized by hamster monoclonal antibodiesD A Croix, H Y Yeh, J Sedlacek, et al.
Virus Research|March 22, 2000
Fusion of uninfected T-cells occurs with immature HIV-1 protease-mutant, but not morphologically similar protease inhibitor derived particlesM K Bahmani, M Kameoka, T Goto, et al.
Virology|February 1, 1993
Myeloblastosis associated virus (MAV) proteinase site-mutated to be HIV-like has a higher activity and allows production of infectious but morphologically altered virusJ Sedlacek, M Fabry, J E Coward, et al.
International Immunology|November 14, 1997
Exposure of resting peripheral blood T cells to HIV-1 particles generates CD25+ killer cells in a small subset, leading to induction of apoptosis in bystander cellsM Kameoka, S Suzuki, T Kimura, et al.
Pageof 11

Showing results (81-90 of 106) with videos related to

Sort By:
Pageof 11
Virology|January 15, 1980
Preparations enriched for "immature" murine leukemia virus particles that remain in tissue culture fluids are deficient in Pr65gag proteolyic activityY Yoshinaka, K Ishigame, T Ohno, et al.
Nucleic Acids Research|November 25, 1989
Sequence of human enteric adenovirus type 41 Tak fiber protein geneN J Pieniazek, S B Slemenda, D Pieniazek, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 1, 1986
Myristylation site in Pr65gag is essential for virus particle formation by Moloney murine leukemia virusA Rein, M R McClure, N R Rice, et al.
Virology|August 1, 1980
Quantitation of the interaction between adenovirus types 2 and 5 and microtubules inside infected cellsB D Miles, R B Luftig, J A Weatherbee, et al.
Virology|January 1, 1990
Differential growth of human enteric adenovirus 41 (TAK) in continuous cell linesD Pieniazek, N J Pieniazek, D Macejak, et al.
International Immunology|November 1, 1996
Induction of apoptosis by protease-defective particle preparations of human immunodeficiency virus type 1 is specific to a subset of U937-derived subclonesM Kameoka, T Kimura, Q Zhong, et al.
Journal of Acquired Immune Deficiency Syndromes|June 1, 1993
A dominant epitope of HIV-1 protease recognized by hamster monoclonal antibodiesD A Croix, H Y Yeh, J Sedlacek, et al.
Virus Research|March 22, 2000
Fusion of uninfected T-cells occurs with immature HIV-1 protease-mutant, but not morphologically similar protease inhibitor derived particlesM K Bahmani, M Kameoka, T Goto, et al.
Virology|February 1, 1993
Myeloblastosis associated virus (MAV) proteinase site-mutated to be HIV-like has a higher activity and allows production of infectious but morphologically altered virusJ Sedlacek, M Fabry, J E Coward, et al.
International Immunology|November 14, 1997
Exposure of resting peripheral blood T cells to HIV-1 particles generates CD25+ killer cells in a small subset, leading to induction of apoptosis in bystander cellsM Kameoka, S Suzuki, T Kimura, et al.
Pageof 11