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Oncogene|February 19, 1998
The PEA3 Ets transcription factor is a downstream target of the HER2/Neu receptor tyrosine kinaseR C O'Hagan, J A HassellOncogene|September 19, 1996
The activity of the Ets transcription factor PEA3 is regulated by two distinct MAPK cascadesR C O'Hagan, R G Tozer, M Symons, et al.Genes & Development|January 20, 2000
Essential role for Max in early embryonic growth and developmentH Shen-Li, R C O'Hagan, H Hou, et al.Genes & Development|September 6, 2000
Myc-enhanced expression of Cul1 promotes ubiquitin-dependent proteolysis and cell cycle progressionR C O'Hagan, M Ohh, G David, et al.Genes & Development|June 3, 2000
N-myc can functionally replace c-myc in murine development, cellular growth, and differentiationB A Malynn, I M de Alboran, R C O'Hagan, et al.Immunity|February 13, 2001
Analysis of C-MYC function in normal cells via conditional gene-targeted mutationI M de Alboran, R C O'Hagan, F Gärtner, et al.Oncogene|February 18, 1999
Telomerase reverse transcriptase gene is a direct target of c-Myc but is not functionally equivalent in cellular transformationR A Greenberg, R C O'Hagan, H Deng, et al.Oncogene|October 31, 1997
HER2/Neu and the Ets transcription activator PEA3 are coordinately upregulated in human breast cancerC C Benz, R C O'Hagan, B Richter, et al.Nature Genetics|February 2, 2000
Gene-target recognition among members of the myc superfamily and implications for oncogenesisR C O'Hagan, N Schreiber-Agus, K Chen, et al.Molecular Cell|January 10, 2002
Impaired nonhomologous end-joining provokes soft tissue sarcomas harboring chromosomal translocations, amplifications, and deletionsN E Sharpless, D O Ferguson, R C O'Hagan, et al.Pageof 1