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Annals of Internal Medicine|October 8, 2008
Systematic review: D-dimer to predict recurrent disease after stopping anticoagulant therapy for unprovoked venous thromboembolismMadeleine Verhovsek, James D Douketis, Qilong Yi, et al.Annals of Internal Medicine|October 20, 2010
Patient-level meta-analysis: effect of measurement timing, threshold, and patient age on ability of D-dimer testing to assess recurrence risk after unprovoked venous thromboembolismJames Douketis, Alberto Tosetto, Maura Marcucci, et al.BMJ (Clinical Research Ed.)|February 26, 2011
Risk of recurrence after venous thromboembolism in men and women: patient level meta-analysisJames Douketis, Alberto Tosetto, Maura Marcucci, et al.Pancreas|March 23, 2005
Use of recombinant activated factor VII for bleeding in pancreatitis: a case seriesMike A Laffan, R Campbell Tait, Jan Blatný, et al.European Journal of Haematology|July 11, 2012
History of non-fatal cardiovascular disease in a cohort of Dutch and British patients with haemophiliaDietje E Fransen van de Putte, Kathelijn Fischer, Michael Makris, et al.Thrombosis and Haemostasis|September 8, 2012
Increased prevalence of hypertension in haemophilia patientsDietje E Fransen van de Putte, Kathelijn Fischer, Michael Makris, et al.Thrombosis and Haemostasis|November 10, 2012
Unfavourable cardiovascular disease risk profiles in a cohort of Dutch and British haemophilia patientsDietje E Fransen van de Putte, Kathelijn Fischer, Michael Makris, et al.Journal of Thrombosis and Haemostasis : JTH|July 8, 2020
A new pedigree with thrombomodulin-associated coagulopathy in which delayed fibrinolysis is partially attenuated by co-inherited TAFI deficiencySarah K Westbury, Claire S Whyte, Jonathan Stephens, et al.The American Journal of Clinical Nutrition|December 8, 2005
Randomized controlled trial of homocysteine-lowering vitamin treatment in elderly patients with vascular diseaseDavid J Stott, Graham MacIntosh, Gordon D O Lowe, et al.British Journal of Haematology|March 10, 2005
The prevalence of the cysteine1584 variant of von Willebrand factor is increased in type 1 von Willebrand disease: co-segregation with increased susceptibility to ADAMTS13 proteolysis but not clinical phenotypeDerrick J Bowen, Peter W Collins, Will Lester, et al.Pageof 3