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Molecular Microbiology|January 14, 1998
Helicobacter pylori toxin VacA induces vacuole formation by acting in the cell cytosolM de Bernard, B Arico, E Papini, et al.
Biophysical Journal|November 27, 2001
How the loop and middle regions influence the properties of Helicobacter pylori VacA channelsF Tombola, C Pagliaccia, S Campello, et al.
Journal of Neurochemistry|December 3, 1999
Botulinum neurotoxin E-insensitive mutants of SNAP-25 fail to bind VAMP but support exocytosisP Washbourne, N Bortoletto, M E Graham, et al.
Journal of Neurochemistry|October 1, 1995
Phosphorylation of VAMP/synaptobrevin in synaptic vesicles by endogenous protein kinasesH B Nielander, F Onofri, F Valtorta, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 11, 1994
Cellular vacuoles induced by Helicobacter pylori originate from late endosomal compartmentsE Papini, M de Bernard, E Milia, et al.
The Journal of Clinical Investigation|August 26, 1998
Selective increase of the permeability of polarized epithelial cell monolayers by Helicobacter pylori vacuolating toxinE Papini, B Satin, N Norais, et al.
Biochemical and Biophysical Research Communications|July 24, 1998
The acid activation of Helicobacter pylori toxin VacA: structural and membrane binding studiesM Molinari, C Galli, M de Bernard, et al.
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