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Protein Science : a Publication of the Protein Society|April 5, 2013
Crystallographic analysis of the ENTH domain from yeast epsin Ent2 that induces a cell division phenotypeGregory T Costakes, Arpita Sen, R Claudio Aguilar, et al.
Communicative & Integrative Biology|April 22, 2011
Epsins' novel role in cancer cell invasionBrian G Coon, Daniel M Direnzo, Stephen F Konieczny, et al.
Human Molecular Genetics|August 25, 2009
Lowe syndrome patient fibroblasts display Ocrl1-specific cell migration defects that cannot be rescued by the homologous Inpp5b phosphataseBrian G Coon, Debarati Mukherjee, Claudia B Hanna, et al.
Journal of Cell Science|July 23, 2020
The Na+ pump Ena1 is a yeast epsin-specific cargo requiring its ubiquitylation and phosphorylation sites for internalizationArpita Sen, Wen-Chieh Hsieh, Claudia B Hanna, et al.
Human Molecular Genetics|May 12, 2020
Lowe syndrome patient cells display mTOR- and RhoGTPase-dependent phenotypes alleviated by rapamycin and statinsKayalvizhi Madhivanan, Swetha Ramadesikan, Wen-Chieh Hsieh, et al.
International Journal of Cancer|September 9, 2011
Fibronectin attachment protein from bacillus Calmette-Guerin as targeting agent for bladder tumor cellsBrian G Coon, Scott Crist, Andrés M González-Bonet, et al.
Human Molecular Genetics|January 10, 2012
The Lowe syndrome protein OCRL1 is involved in primary cilia assemblyBrian G Coon, Victor Hernandez, Kayalvizhi Madhivanan, et al.
Human Molecular Genetics|January 31, 2021
Genotype & phenotype in Lowe Syndrome: specific OCRL1 patient mutations differentially impact cellular phenotypesSwetha Ramadesikan, Lisette Skiba, Jennifer Lee, et al.
Journal of Cell Science|June 18, 2009
The yeast endocytic protein Epsin 2 functions in a cell-division signaling pathwayDebarati Mukherjee, Brian G Coon, Daniel F Edwards, et al.
Journal of Cell Science|August 15, 2013
Bem3, a Cdc42 GTPase-activating protein, traffics to an intracellular compartment and recruits the secretory Rab GTPase Sec4 to endomembranesDebarati Mukherjee, Arpita Sen, Douglas R Boettner, et al.
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