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R Cummins

Showing results (121-130 of 319) with videos related to

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The Journal of Physiology|July 5, 2008
Paroxysmal extreme pain disorder mutations within the D3/S4-S5 linker of Nav1.7 cause moderate destabilization of fast inactivationBrian W Jarecki, Patrick L Sheets, James O Jackson, et al.
Novartis Foundation Symposium|January 5, 2002
Diverse functions and dynamic expression of neuronal sodium channelsStephen G Waxman, Theodore R Cummins, Joel A Black, et al.
Journal of Neurophysiology|February 26, 1999
In vivo NGF deprivation reduces SNS expression and TTX-R sodium currents in IB4-negative DRG neuronsJ Fjell, T R Cummins, K Fried, et al.
Progress in Brain Research|December 1, 2000
Sodium channels and the molecular pathophysiology of painT R Cummins, S D Dib-Hajj, J A Black, et al.
Trends in Neurosciences|October 24, 2007
From genes to pain: Na v 1.7 and human pain disordersSulayman D Dib-Hajj, Theodore R Cummins, Joel A Black, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 19, 1995
Serine-1321-independent regulation of the mu 1 adult skeletal muscle Na+ channel by protein kinase CS Bendahhou, T R Cummins, J F Potts, et al.
Scientific Reports|August 15, 2013
Structural basis for the modulation of the neuronal voltage-gated sodium channel NaV1.6 by calmodulinVishnu Priyanka Reddy Chichili, Yucheng Xiao, J Seetharaman, et al.
Frontiers in Pharmacology|May 7, 2019
A Review of the Therapeutic Potential of Recently Developed G Protein-Biased Kappa AgonistsKendall L Mores, Benjamin R Cummins, Robert J Cassell, et al.
Biomedicines|September 27, 2025
Voltage-Gated Sodium Channel Dysfunction in Epilepsy: Zebrafish Models for TherapeuticsAngela Gyamfi, Priyadharshini Manikandan, William A Cisneros, et al.
Proceedings of the Mine Medical Officers' Association of S. A|May 1, 1975
Enzyme patterns in primary human liver cancersE Cayanis, R R Cummins, K D Hammond, et al.
Pageof 32

Showing results (121-130 of 319) with videos related to

Sort By:
Pageof 32
The Journal of Physiology|July 5, 2008
Paroxysmal extreme pain disorder mutations within the D3/S4-S5 linker of Nav1.7 cause moderate destabilization of fast inactivationBrian W Jarecki, Patrick L Sheets, James O Jackson, et al.
Novartis Foundation Symposium|January 5, 2002
Diverse functions and dynamic expression of neuronal sodium channelsStephen G Waxman, Theodore R Cummins, Joel A Black, et al.
Journal of Neurophysiology|February 26, 1999
In vivo NGF deprivation reduces SNS expression and TTX-R sodium currents in IB4-negative DRG neuronsJ Fjell, T R Cummins, K Fried, et al.
Progress in Brain Research|December 1, 2000
Sodium channels and the molecular pathophysiology of painT R Cummins, S D Dib-Hajj, J A Black, et al.
Trends in Neurosciences|October 24, 2007
From genes to pain: Na v 1.7 and human pain disordersSulayman D Dib-Hajj, Theodore R Cummins, Joel A Black, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 19, 1995
Serine-1321-independent regulation of the mu 1 adult skeletal muscle Na+ channel by protein kinase CS Bendahhou, T R Cummins, J F Potts, et al.
Scientific Reports|August 15, 2013
Structural basis for the modulation of the neuronal voltage-gated sodium channel NaV1.6 by calmodulinVishnu Priyanka Reddy Chichili, Yucheng Xiao, J Seetharaman, et al.
Frontiers in Pharmacology|May 7, 2019
A Review of the Therapeutic Potential of Recently Developed G Protein-Biased Kappa AgonistsKendall L Mores, Benjamin R Cummins, Robert J Cassell, et al.
Biomedicines|September 27, 2025
Voltage-Gated Sodium Channel Dysfunction in Epilepsy: Zebrafish Models for TherapeuticsAngela Gyamfi, Priyadharshini Manikandan, William A Cisneros, et al.
Proceedings of the Mine Medical Officers' Association of S. A|May 1, 1975
Enzyme patterns in primary human liver cancersE Cayanis, R R Cummins, K D Hammond, et al.
Pageof 32