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R Cummins

Showing results (201-210 of 319) with videos related to

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Neuroreport|May 28, 1998
SNS Na+ channel expression increases in dorsal root ganglion neurons in the carrageenan inflammatory pain modelM Tanaka, T R Cummins, K Ishikawa, et al.
Brain Research Reviews|January 20, 2009
Voltage-gated sodium channels in pain states: role in pathophysiology and targets for treatmentSulayman D Dib-Hajj, Alexander M Binshtok, Theodore R Cummins, et al.
The Journal of Biological Chemistry|January 19, 2012
Calcium/calmodulin-dependent protein kinase II (CaMKII) inhibition induces neurotoxicity via dysregulation of glutamate/calcium signaling and hyperexcitabilityNicole M Ashpole, Weihua Song, Tatiana Brustovetsky, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 9, 2019
Increased Resurgent Sodium Currents in Nav1.8 Contribute to Nociceptive Sensory Neuron Hyperexcitability Associated with Peripheral NeuropathiesYucheng Xiao, Cindy Barbosa, Zifan Pei, et al.
Molecular and Cellular Neurosciences|March 31, 2005
CAP-1A is a novel linker that binds clathrin and the voltage-gated sodium channel Na(v)1.8Chuanju Liu, Theodore R Cummins, Lynda Tyrrell, et al.
JMIR Formative Research|March 11, 2025
Telemedicine Prescribing by US Mental Health Care Providers: National Cross-Sectional SurveyMollie R Cummins, Julia Ivanova, Hiral Soni, et al.
Journal of Medical Internet Research|January 23, 2025
Regulation and Compliance in Telemedicine: ViewpointJulia Ivanova, Mollie R Cummins, Triton Ong, et al.
Learning Health Systems|January 21, 2026
Exploring Factors That Impact Genetic Counseling Referral and Uptake Using Learning Health ApproachesSamantha Greenberg, Bob Wong, Katherine A Sward, et al.
Journal of Neurophysiology|August 7, 2002
GDNF and NGF reverse changes in repriming of TTX-sensitive Na(+) currents following axotomy of dorsal root ganglion neuronsAndreas Leffler, Theodore R Cummins, Sulayman D Dib-Hajj, et al.
Neuron|April 1, 1993
Functional consequences of a Na+ channel mutation causing hyperkalemic periodic paralysisT R Cummins, J Zhou, F J Sigworth, et al.
Pageof 32

Showing results (201-210 of 319) with videos related to

Sort By:
Pageof 32
Neuroreport|May 28, 1998
SNS Na+ channel expression increases in dorsal root ganglion neurons in the carrageenan inflammatory pain modelM Tanaka, T R Cummins, K Ishikawa, et al.
Brain Research Reviews|January 20, 2009
Voltage-gated sodium channels in pain states: role in pathophysiology and targets for treatmentSulayman D Dib-Hajj, Alexander M Binshtok, Theodore R Cummins, et al.
The Journal of Biological Chemistry|January 19, 2012
Calcium/calmodulin-dependent protein kinase II (CaMKII) inhibition induces neurotoxicity via dysregulation of glutamate/calcium signaling and hyperexcitabilityNicole M Ashpole, Weihua Song, Tatiana Brustovetsky, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 9, 2019
Increased Resurgent Sodium Currents in Nav1.8 Contribute to Nociceptive Sensory Neuron Hyperexcitability Associated with Peripheral NeuropathiesYucheng Xiao, Cindy Barbosa, Zifan Pei, et al.
Molecular and Cellular Neurosciences|March 31, 2005
CAP-1A is a novel linker that binds clathrin and the voltage-gated sodium channel Na(v)1.8Chuanju Liu, Theodore R Cummins, Lynda Tyrrell, et al.
JMIR Formative Research|March 11, 2025
Telemedicine Prescribing by US Mental Health Care Providers: National Cross-Sectional SurveyMollie R Cummins, Julia Ivanova, Hiral Soni, et al.
Journal of Medical Internet Research|January 23, 2025
Regulation and Compliance in Telemedicine: ViewpointJulia Ivanova, Mollie R Cummins, Triton Ong, et al.
Learning Health Systems|January 21, 2026
Exploring Factors That Impact Genetic Counseling Referral and Uptake Using Learning Health ApproachesSamantha Greenberg, Bob Wong, Katherine A Sward, et al.
Journal of Neurophysiology|August 7, 2002
GDNF and NGF reverse changes in repriming of TTX-sensitive Na(+) currents following axotomy of dorsal root ganglion neuronsAndreas Leffler, Theodore R Cummins, Sulayman D Dib-Hajj, et al.
Neuron|April 1, 1993
Functional consequences of a Na+ channel mutation causing hyperkalemic periodic paralysisT R Cummins, J Zhou, F J Sigworth, et al.
Pageof 32