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The Journal of Physiology|June 1, 1984
A mutation that alters properties of the calcium channel in Paramecium tetraureliaR D Hinrichsen, Y SaimiGene|September 15, 1994
The identification of a complex family of low-molecular-weight GTP-binding protein homologues from Paramecium tetraurelia by PCR cloningD Fraga, R D HinrichsenProceedings of the National Academy of Sciences of the United States of America|February 15, 1993
Regulation of peptide-calmodulin complexes by protein kinase C in vivoR D Hinrichsen, P J BlackshearGenetics|November 1, 1984
Mutants with altered Ca2+-channel properties in Paramecium tetraurelia: isolation, characterization and genetic analysisR D Hinrichsen, Y Saimi, C KungNucleic Acids Research|April 11, 1994
Extremely short 20-33 nucleotide introns are the standard length in Paramecium tetraureliaC B Russell, D Fraga, R D HinrichsenProceedings of the National Academy of Sciences of the United States of America|September 15, 1992
3'-modified antisense oligodeoxyribonucleotides complementary to calmodulin mRNA alter behavioral responses in ParameciumR D Hinrichsen, D Fraga, M W ReedJournal of Neurogenetics|September 1, 1985
A single gene mutation that affects a potassium conductance and resting membrane potential in ParameciumE A Richard, R D Hinrichsen, C KungGenetics|November 1, 1991
An intragenic suppressor of a calmodulin mutation in Paramecium: genetic and biochemical characterizationR D Hinrichsen, M Pollock, T Hennessey, et al.Cell Motility|January 1, 1984
Mutants in paramecium tetraurelia defective in their axonemal response to calciumR D Hinrichsen, Y Saimi, T Hennessey, et al.Proceedings of the National Academy of Sciences of the United States of America|August 1, 1983
Mutant analysis shows that the Ca2+-induced K+ current shuts off one type of excitation in ParameciumY Saimi, R D Hinrichsen, M Forte, et al.Pageof 2