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R Demling

Showing results (21-30 of 57) with videos related to

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The Journal of Burn Care & Rehabilitation|December 24, 1997
Plasma catalase and glutathione levels are decreased in response to inhalation injuryC LaLonde, U Nayak, J Hennigan, et al.
The Journal of Burn Care & Rehabilitation|October 28, 1998
Development of a burn rehabilitation unit: impact on burn center length of stay and functional outcomeL DeSanti, L Lincoln, F Egan, et al.
The Journal of Burn Care & Rehabilitation|September 1, 1996
Antioxidants prevent the cellular deficit produced in response to burn injuryC LaLonde, U Nayak, J Hennigan, et al.
Circulatory Shock|January 1, 1994
Lung and systemic oxidant and antioxidant activity after graded smoke exposure in the ratC Lalonde, L Picard, C Campbell, et al.
Archives of Surgery (Chicago, Ill. : 1960)|November 1, 1988
Endotoxin causes hydrogen peroxide-induced lung lipid peroxidation and prostanoid productionR Demling, C Lalonde, A Seekamp, et al.
Current Problems in Surgery|April 1, 1993
Multiple-organ dysfunction in the surgical patient: pathophysiology, prevention, and treatmentR Demling, C LaLonde, P Saldinger, et al.
Shock (Augusta, Ga.)|September 1, 1994
Comparison between lung and liver lipid peroxidation and mortality after zymosan peritonitis in the ratR Demling, U Nayak, K Ikegami, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|July 1, 1994
Smoke inhalation injury in sheep is caused by the particle phase, not the gas phaseC Lalonde, R Demling, J Brain, et al.
Shock (Augusta, Ga.)|February 1, 1994
Changes in lung and systemic oxidant and antioxidant activity after smoke inhalationR Demling, C Lalonde, L Picard, et al.
Critical Care Medicine|December 1, 1993
Relationship of burn-induced lung lipid peroxidation on the degree of injury after smoke inhalation and a body burnR Demling, L Picard, C Campbell, et al.
Pageof 6

Showing results (21-30 of 57) with videos related to

Sort By:
Pageof 6
The Journal of Burn Care & Rehabilitation|December 24, 1997
Plasma catalase and glutathione levels are decreased in response to inhalation injuryC LaLonde, U Nayak, J Hennigan, et al.
The Journal of Burn Care & Rehabilitation|October 28, 1998
Development of a burn rehabilitation unit: impact on burn center length of stay and functional outcomeL DeSanti, L Lincoln, F Egan, et al.
The Journal of Burn Care & Rehabilitation|September 1, 1996
Antioxidants prevent the cellular deficit produced in response to burn injuryC LaLonde, U Nayak, J Hennigan, et al.
Circulatory Shock|January 1, 1994
Lung and systemic oxidant and antioxidant activity after graded smoke exposure in the ratC Lalonde, L Picard, C Campbell, et al.
Archives of Surgery (Chicago, Ill. : 1960)|November 1, 1988
Endotoxin causes hydrogen peroxide-induced lung lipid peroxidation and prostanoid productionR Demling, C Lalonde, A Seekamp, et al.
Current Problems in Surgery|April 1, 1993
Multiple-organ dysfunction in the surgical patient: pathophysiology, prevention, and treatmentR Demling, C LaLonde, P Saldinger, et al.
Shock (Augusta, Ga.)|September 1, 1994
Comparison between lung and liver lipid peroxidation and mortality after zymosan peritonitis in the ratR Demling, U Nayak, K Ikegami, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|July 1, 1994
Smoke inhalation injury in sheep is caused by the particle phase, not the gas phaseC Lalonde, R Demling, J Brain, et al.
Shock (Augusta, Ga.)|February 1, 1994
Changes in lung and systemic oxidant and antioxidant activity after smoke inhalationR Demling, C Lalonde, L Picard, et al.
Critical Care Medicine|December 1, 1993
Relationship of burn-induced lung lipid peroxidation on the degree of injury after smoke inhalation and a body burnR Demling, L Picard, C Campbell, et al.
Pageof 6