R E Gale|H Wheadon|P Boulos|D C Linch - JoVE Visualize

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Leukemia|February 1, 1996

Quantification of X-chromosome inactivation patterns in haematological samples using the DNA PCR-based HUMARA assayR E Gale, C A Mein, D C Linch

Leukemia|November 21, 1998

Quantification of X-chromosome inactivation patterns using RT-PCR of the polymorphic iduronate-2-sulphatase gene and correlation of the results obtained with DNA-based techniquesC N Harrison, R E Gale, D C Linch

British Journal of Haematology|October 29, 1998

Activating point mutations in the betaC chain of the GM-CSF, IL-3 and IL-5 receptors are not a major contributory factor in the pathogenesis of acute myeloid leukaemiaR W Freeburn, R E Gale, D C Linch

Leukemia|February 11, 2005

PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB, Mapkinase and p53 pathwaysV L Grandage, R E Gale, D C Linch, et al.

Leukemia|April 1, 1996

Variable expression of p16 protein in patients with acute myeloid leukemia without gross rearrangements at the DNA levelR Jamal, N S Thomas, R E Gale, et al.

British Journal of Haematology|May 12, 1998

Mutations of the granulocyte-colony stimulating factor receptor in patients with severe congenital neutropenia are not required for transformation to acute myeloid leukaemia and may be a bystander phenomenonT Bernard, R E Gale, J P Evans, et al.

British Journal of Haematology|October 23, 1997

Acquired skewing of X-chromosome inactivation patterns in myeloid cells of the elderly suggests stochastic clonal loss with ageR E Gale, A K Fielding, C N Harrison, et al.

Experimental Hematology|April 1, 1997

Analysis of the coding sequence for the GM-CSF receptor alpha and beta chains in patients with juvenile chronic myeloid leukemia (JCML)R W Freeburn, R E Gale, H M Wagner, et al.

Blood|January 13, 1999

A large proportion of patients with a diagnosis of essential thrombocythemia do not have a clonal disorder and may be at lower risk of thrombotic complicationsC N Harrison, R E Gale, S J Machin, et al.

Leukemia|January 1, 1996

The beta subunit common to the GM-CSF, IL-3 and IL-5 receptors is highly polymorphic but pathogenic point mutations in patients with acute myeloid leukaemia (AML) are rareR W Freeburn, R E Gale, H M Wagner, et al.

Pageof 28

Showing results (11-20 of 278) with videos related to

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Pageof 28

Leukemia|February 1, 1996

Quantification of X-chromosome inactivation patterns in haematological samples using the DNA PCR-based HUMARA assayR E Gale, C A Mein, D C Linch

Leukemia|November 21, 1998

Quantification of X-chromosome inactivation patterns using RT-PCR of the polymorphic iduronate-2-sulphatase gene and correlation of the results obtained with DNA-based techniquesC N Harrison, R E Gale, D C Linch

British Journal of Haematology|October 29, 1998

Activating point mutations in the betaC chain of the GM-CSF, IL-3 and IL-5 receptors are not a major contributory factor in the pathogenesis of acute myeloid leukaemiaR W Freeburn, R E Gale, D C Linch

Leukemia|February 11, 2005

PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB, Mapkinase and p53 pathwaysV L Grandage, R E Gale, D C Linch, et al.

Leukemia|April 1, 1996

Variable expression of p16 protein in patients with acute myeloid leukemia without gross rearrangements at the DNA levelR Jamal, N S Thomas, R E Gale, et al.

British Journal of Haematology|May 12, 1998

Mutations of the granulocyte-colony stimulating factor receptor in patients with severe congenital neutropenia are not required for transformation to acute myeloid leukaemia and may be a bystander phenomenonT Bernard, R E Gale, J P Evans, et al.

British Journal of Haematology|October 23, 1997

Acquired skewing of X-chromosome inactivation patterns in myeloid cells of the elderly suggests stochastic clonal loss with ageR E Gale, A K Fielding, C N Harrison, et al.

Experimental Hematology|April 1, 1997

Analysis of the coding sequence for the GM-CSF receptor alpha and beta chains in patients with juvenile chronic myeloid leukemia (JCML)R W Freeburn, R E Gale, H M Wagner, et al.

Blood|January 13, 1999

A large proportion of patients with a diagnosis of essential thrombocythemia do not have a clonal disorder and may be at lower risk of thrombotic complicationsC N Harrison, R E Gale, S J Machin, et al.

Leukemia|January 1, 1996

The beta subunit common to the GM-CSF, IL-3 and IL-5 receptors is highly polymorphic but pathogenic point mutations in patients with acute myeloid leukaemia (AML) are rareR W Freeburn, R E Gale, H M Wagner, et al.

Pageof 28