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R E Gale

Showing results (11-20 of 55) with videos related to

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The British Journal of Ophthalmology|October 1, 1980
Oxygen dissociation curve in Eales's diseaseD L Boase, R E Gale, E R Huehns
Leukemia|February 1, 1996
Quantification of X-chromosome inactivation patterns in haematological samples using the DNA PCR-based HUMARA assayR E Gale, C A Mein, D C Linch
Leukemia|November 21, 1998
Quantification of X-chromosome inactivation patterns using RT-PCR of the polymorphic iduronate-2-sulphatase gene and correlation of the results obtained with DNA-based techniquesC N Harrison, R E Gale, D C Linch
British Journal of Haematology|October 29, 1998
Activating point mutations in the betaC chain of the GM-CSF, IL-3 and IL-5 receptors are not a major contributory factor in the pathogenesis of acute myeloid leukaemiaR W Freeburn, R E Gale, D C Linch
Nature|July 12, 1979
Human embryonic haemoglobins Gower 1 and Gower 2R E Gale, J B Clegg, E R Huehns
Advances in Experimental Medicine and Biology|July 4, 1977
Oxygen affinity changes in the red cells of embryonic and neonatal miceR E Gale, R M Wells, E R Huehns
Lancet (London, England)|May 17, 1997
Glutathione S-transferase theta 1 (GSTT1) gene defect in myelodysplasia and acute myeloid leukaemiaT Basu, R E Gale, S Langabeer, et al.
Blood|May 15, 1994
Tissue specificity of X-chromosome inactivation patternsR E Gale, H Wheadon, P Boulos, et al.
Leukemia|February 11, 2005
PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB, Mapkinase and p53 pathwaysV L Grandage, R E Gale, D C Linch, et al.
Leukemia|October 16, 2021
Correction: PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kB, MAPkinase and p53 pathwaysV L Grandage, R E Gale, D C Linch, et al.
Pageof 6

Showing results (11-20 of 55) with videos related to

Sort By:
Pageof 6
The British Journal of Ophthalmology|October 1, 1980
Oxygen dissociation curve in Eales's diseaseD L Boase, R E Gale, E R Huehns
Leukemia|February 1, 1996
Quantification of X-chromosome inactivation patterns in haematological samples using the DNA PCR-based HUMARA assayR E Gale, C A Mein, D C Linch
Leukemia|November 21, 1998
Quantification of X-chromosome inactivation patterns using RT-PCR of the polymorphic iduronate-2-sulphatase gene and correlation of the results obtained with DNA-based techniquesC N Harrison, R E Gale, D C Linch
British Journal of Haematology|October 29, 1998
Activating point mutations in the betaC chain of the GM-CSF, IL-3 and IL-5 receptors are not a major contributory factor in the pathogenesis of acute myeloid leukaemiaR W Freeburn, R E Gale, D C Linch
Nature|July 12, 1979
Human embryonic haemoglobins Gower 1 and Gower 2R E Gale, J B Clegg, E R Huehns
Advances in Experimental Medicine and Biology|July 4, 1977
Oxygen affinity changes in the red cells of embryonic and neonatal miceR E Gale, R M Wells, E R Huehns
Lancet (London, England)|May 17, 1997
Glutathione S-transferase theta 1 (GSTT1) gene defect in myelodysplasia and acute myeloid leukaemiaT Basu, R E Gale, S Langabeer, et al.
Blood|May 15, 1994
Tissue specificity of X-chromosome inactivation patternsR E Gale, H Wheadon, P Boulos, et al.
Leukemia|February 11, 2005
PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB, Mapkinase and p53 pathwaysV L Grandage, R E Gale, D C Linch, et al.
Leukemia|October 16, 2021
Correction: PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kB, MAPkinase and p53 pathwaysV L Grandage, R E Gale, D C Linch, et al.
Pageof 6