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Neuroscience Letters|March 28, 1994
Activities of monoamine oxidase-A and -B are altered in the brains of congenitally hyperammonemic sparse-fur (spf) miceV L Rao, I A Qureshi, R F ButterworthClinical Biochemistry|October 1, 1975
A reliable method for the simultaneous estimation of dopamine, noradrenaline and serotonin in discrete areas of brainR F Butterworth, F Landreville, M Guitard, et al.Neurochemical Research|April 1, 1986
Activities of thiamine-dependent enzymes in two experimental models of thiamine-deficiency encephalopathy. 2. alpha-Ketoglutarate dehydrogenaseR F Butterworth, J F Giguère, A M BesnardBrain Research|August 8, 1997
Increased neuronal nitric oxide synthase expression in brain following portacaval anastomosisV L Rao, R M Audet, R F ButterworthBrain Research|December 24, 1993
Decreased activities of thiamine diphosphatase in frontal and temporal cortex in Alzheimer's diseaseV L Rao, J S Richardson, R F ButterworthThe Canadian Journal of Neurological Sciences. Le Journal Canadien Des Sciences Neurologiques|February 1, 1978
Effect of alloxan diabetes on cerebellar amino acidsR F Butterworth, E Hamel, F Landreville, et al.Pediatric Research|December 1, 1993
Increased densities of binding sites for the peripheral-type benzodiazepine receptor ligand [3H]PK 11195 in congenital ornithine transcarbamylase-deficient sparse fur mouseV L Rao, I A Qureshi, R F ButterworthHepatology (Baltimore, Md.)|March 1, 1997
Protective effect of L-carnitine in ammonia-precipitated encephalopathy in the portacaval shunted ratG Therrien, C Rose, J Butterworth, et al.European Journal of Pharmacology|December 19, 1996
Vesicular dysfunction during experimental thiamine deficiency is indicated by alterations in dopamine metabolismD D Mousseau, V L Rao, R F ButterworthNeurochemical Research|October 1, 1995
Loss of [3H]kainate and of NMDA-displaceable [3H]glutamate binding sites in brain in thiamine deficiency: results of a quantitative autoradiographic studyC Peterson, M Héroux, J Lavoie, et al.Pageof 28