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Proceedings of the National Academy of Sciences of the United States of America|February 1, 1988
"Calciosome," a cytoplasmic organelle: the inositol 1,4,5-trisphosphate-sensitive Ca2+ store of nonmuscle cells?P Volpe, K H Krause, S Hashimoto, et al.Biochemical and Biophysical Research Communications|May 29, 1986
PDGF-induced receptor phosphorylation and phosphoinositide hydrolysis are unaffected by protein kinase C activation in mouse Swiss 3T3 and human skin fibroblastsE Sturani, L M Vicentini, R Zippel, et al.The Journal of Cell Biology|August 1, 1995
Overexpression of calreticulin increases the Ca2+ capacity of rapidly exchanging Ca2+ stores and reveals aspects of their lumenal microenvironment and functionC Bastianutto, E Clementi, F Codazzi, et al.The Journal of Cell Biology|February 1, 1995
Differential localization and functional role of calsequestrin in growing and differentiated myoblastsM Raichman, M C Panzeri, E Clementi, et al.The Journal of Biological Chemistry|May 21, 1999
The p75(NTR)-induced apoptotic program develops through a ceramide-caspase pathway negatively regulated by nitric oxideJ P Lièvremont, C Sciorati, E Morandi, et al.The Journal of Biological Chemistry|October 25, 1991
Intracellular Ca2+ pools in PC12 cells. A unique, rapidly exchanging pool is sensitive to both inositol 1,4,5-trisphosphate and caffeine-ryanodineD Zacchetti, E Clementi, C Fasolato, et al.The Biochemical Journal|October 15, 1990
Calreticulin is a candidate for a calsequestrin-like function in Ca2(+)-storage compartments (calciosomes) of liver and brainS Treves, M De Mattei, M Landfredi, et al.Neuroscience|July 22, 2009
Analysis of pre- and postsynaptic activity in the frog semicircular canal following ototoxic insult: differential recovery of background and evoked afferent activityG Russo, D Calzi, L Gioglio, et al.Journal of Cardiovascular Pharmacology|January 1, 1988
Immunocytological identification of the microsomal calcium store of nonmuscle cellsT Pozzan, P Volpe, F Zorzato, et al.Proceedings of the National Academy of Sciences of the United States of America|May 3, 2000
Nitric oxide inhibits tumor necrosis factor-alpha-induced apoptosis by reducing the generation of ceramideC De Nadai, P Sestili, O Cantoni, et al.Pageof 22