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Physiological Genomics|October 28, 2010
Delineating the angiogenic gene expression profile before pulmonary vascular remodeling in a lamb model of congenital heart diseaseJing Tian, Sohrab Fratz, Yali Hou, et al.
Pulmonary Circulation|April 15, 2024
A single institution anesthetic experience with catheterization of pediatric pulmonary hypertension patientsEmily Morell, Elizabeth Colglazier, Jasmine Becerra, et al.
American Journal of Physiology. Heart and Circulatory Physiology|September 4, 2016
Pulmonary artery smooth muscle cell hyperproliferation and metabolic shift triggered by pulmonary overcirculationJason Boehme, Xutong Sun, Kathryn V Tormos, et al.
Antioxidants & Redox Signaling|December 19, 2012
Disruption of endothelial cell mitochondrial bioenergetics in lambs with increased pulmonary blood flowXutong Sun, Shruti Sharma, Sohrab Fratz, et al.
The Journal of Biological Chemistry|February 25, 2023
Nitration-mediated activation of the small GTPase RhoA stimulates cellular glycolysis through enhanced mitochondrial fissionQing Lu, Xutong Sun, Manivannan Yegambaram, et al.
Pulmonary Circulation|June 4, 2020
Mechanical forces alter endothelin-1 signaling: comparative ovine models of congenital heart diseaseTerry Zhu, Samuel Chiacchia, Rebecca J Kameny, et al.
The New England Journal of Medicine|February 27, 1997
Inhaled nitric oxide and persistent pulmonary hypertension of the newborn. The Inhaled Nitric Oxide Study GroupJ D Roberts, J R Fineman, F C Morin, et al.
Scientific Reports|May 10, 2025
Mechanotransductive stabilization of HIF-1α is inhibited by mitochondrial antioxidant therapy in the setting of pulmonary overcirculationJason T Boehme, Sanjeev A Datar, Xutong Sun, et al.
American Journal of Respiratory Cell and Molecular Biology|January 8, 2014
Endothelin-1 induces a glycolytic switch in pulmonary arterial endothelial cells via the mitochondrial translocation of endothelial nitric oxide synthaseXutong Sun, Sanjiv Kumar, Shruti Sharma, et al.
Redox Biology|November 22, 2020
Activation of the mechanosensitive Ca<sup>2+</sup> channel TRPV4 induces endothelial barrier permeability via the disruption of mitochondrial bioenergeticsQing Lu, Evgeny A Zemskov, Xutong Sun, et al.
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