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The American Journal of Pathology|March 1, 1985
Patterns of pulmonary structural remodeling after experimental paraquat toxicity. The morphogenesis of intraalveolar fibrosisY Fukuda, V J Ferrans, C I Schoenberger, et al.
The American Review of Respiratory Disease|January 1, 1988
Colchicine suppresses the release of fibroblast growth factors from alveolar macrophages in vitro. The basis of a possible therapeutic approach ot the fibrotic disordersS I Rennard, P B Bitterman, T Ozaki, et al.
The American Review of Respiratory Disease|December 1, 1987
Characterization of the lower respiratory tract inflammation of nonsmoking individuals with interstitial lung disease associated with chronic inhalation of inorganic dustsW N Rom, P B Bitterman, S I Rennard, et al.
The American Review of Respiratory Disease|September 1, 1984
Alveolar macrophages in idiopathic pulmonary fibrosis have glucocorticoid receptors, but glucocorticoid therapy does not suppress alveolar macrophage release of fibronectin and alveolar macrophage derived growth factorJ G Lacronique, S I Rennard, P B Bitterman, et al.
Clinical and Experimental Immunology|October 1, 1983
Fibronectin mediates cell attachment to C1q: a mechanism for the localization of fibrosis in inflammatory diseaseS I Rennard, Y F Chen, R A Robbins, et al.
The Journal of Clinical Investigation|October 1, 1981
Antielastases of the human alveolar structures. Implications for the protease-antiprotease theory of emphysemaJ E Gadek, G A Fells, R L Zimmerman, et al.
The Journal of Clinical Investigation|December 1, 1984
Normal human alveolar macrophages obtained by bronchoalveolar lavage have a limited capacity to release interleukin-1M D Wewers, S I Rennard, A J Hance, et al.
The American Review of Respiratory Disease|April 1, 1985
Evidence for chronic inflammation as a component of the interstitial lung disease associated with progressive systemic sclerosisG A Rossi, P B Bitterman, S I Rennard, et al.
The American Review of Respiratory Disease|January 1, 1984
Paraquat-induced pulmonary fibrosis. Role of the alveolitis in modulating the development of fibrosisC I Schoenberger, S I Rennard, P B Bitterman, et al.
Biochemistry|March 25, 1986
Human type III collagen gene expression is coordinately modulated with the type I collagen genes during fibroblast growthM Miskulin, R Dalgleish, B Kluve-Beckerman, et al.
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