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R G Giffard

Showing results (11-20 of 41) with videos related to

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The Journal of Cell Biology|May 1, 1984
Ca2+-dependent binding of severin to actin: a one-to-one complex is formedR G Giffard, A G Weeds, J A Spudich
Science (New York, N.Y.)|June 4, 1993
Neuroprotective effects of glutamate antagonists and extracellular acidityD A Kaku, R G Giffard, D W Choi
British Journal of Neurosurgery|February 24, 2001
Principles of gene therapy: potential applications in the treatment of cerebral ischaemiaM C Papadopoulos, R G Giffard, B A Bell
Neurological Research|February 1, 1996
Mechanism of heat shock protein 72 induction in primary cultured astrocytes after oxygen-glucose deprivationM Bergeron, N F Mivechi, A J Giaccia, et al.
The Journal of Biological Chemistry|April 25, 1997
Increased production of extracellular glutamate by the mitochondrial glutaminase following neuronal deathR Newcomb, X Sun, L Taylor, et al.
Neuroreport|May 14, 1998
Acidosis reduces neuronal apoptosisL Xu, A J Glassford, A J Giaccia, et al.
The European Journal of Neuroscience|September 28, 1998
Potentiation of murine astrocyte antioxidant defence by bcl-2: protection in part reflects elevated glutathione levelsM C Papadopoulos, I L Koumenis, L Xu, et al.
Anesthesiology|October 16, 1999
Overexpression of bcl-xL protects astrocytes from glucose deprivation and is associated with higher glutathione, ferritin, and iron levelsL Xu, I L Koumenis, J L Tilly, et al.
Brain Research|January 8, 1990
Acidosis reduces NMDA receptor activation, glutamate neurotoxicity, and oxygen-glucose deprivation neuronal injury in cortical culturesR G Giffard, H Monyer, C W Christine, et al.
Brain Research|February 14, 1997
Vulnerability to glucose deprivation injury correlates with glutathione levels in astrocytesM C Papadopoulos, I L Koumenis, L L Dugan, et al.
Pageof 5

Showing results (11-20 of 41) with videos related to

Sort By:
Pageof 5
The Journal of Cell Biology|May 1, 1984
Ca2+-dependent binding of severin to actin: a one-to-one complex is formedR G Giffard, A G Weeds, J A Spudich
Science (New York, N.Y.)|June 4, 1993
Neuroprotective effects of glutamate antagonists and extracellular acidityD A Kaku, R G Giffard, D W Choi
British Journal of Neurosurgery|February 24, 2001
Principles of gene therapy: potential applications in the treatment of cerebral ischaemiaM C Papadopoulos, R G Giffard, B A Bell
Neurological Research|February 1, 1996
Mechanism of heat shock protein 72 induction in primary cultured astrocytes after oxygen-glucose deprivationM Bergeron, N F Mivechi, A J Giaccia, et al.
The Journal of Biological Chemistry|April 25, 1997
Increased production of extracellular glutamate by the mitochondrial glutaminase following neuronal deathR Newcomb, X Sun, L Taylor, et al.
Neuroreport|May 14, 1998
Acidosis reduces neuronal apoptosisL Xu, A J Glassford, A J Giaccia, et al.
The European Journal of Neuroscience|September 28, 1998
Potentiation of murine astrocyte antioxidant defence by bcl-2: protection in part reflects elevated glutathione levelsM C Papadopoulos, I L Koumenis, L Xu, et al.
Anesthesiology|October 16, 1999
Overexpression of bcl-xL protects astrocytes from glucose deprivation and is associated with higher glutathione, ferritin, and iron levelsL Xu, I L Koumenis, J L Tilly, et al.
Brain Research|January 8, 1990
Acidosis reduces NMDA receptor activation, glutamate neurotoxicity, and oxygen-glucose deprivation neuronal injury in cortical culturesR G Giffard, H Monyer, C W Christine, et al.
Brain Research|February 14, 1997
Vulnerability to glucose deprivation injury correlates with glutathione levels in astrocytesM C Papadopoulos, I L Koumenis, L L Dugan, et al.
Pageof 5