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BMC Research Notes|August 14, 2015
Genotype and phenotype correlations in Iranian patients with hyperinsulinaemic hypoglycaemiaSenthil Senniappan, Atefeh Sadeghizadeh, Sarah E Flanagan, et al.
Telemedicine Journal and E-Health : the Official Journal of the American Telemedicine Association|August 25, 2021
Veterans Hospital Administration Telehealth Utilization for Recreation and Creative Arts Therapies: A Brief ReportKristin M Story, Mindy E Flanagan, Heather A Brown, et al.
European Journal of Endocrinology|June 1, 2012
Amino acid properties may be useful in predicting clinical outcome in patients with Kir6.2 neonatal diabetesClementine S Fraser, Oscar Rubio-Cabezas, Jennifer A Littlechild, et al.
Pediatric Diabetes|January 8, 2013
Thiamine responsive megaloblastic anemia: a novel SLC19A2 compound heterozygous mutation in two siblingsEnza Mozzillo, Daniela Melis, Mariateresa Falco, et al.
Diabetes|December 11, 2012
GATA6 mutations cause a broad phenotypic spectrum of diabetes from pancreatic agenesis to adult-onset diabetes without exocrine insufficiencyElisa De Franco, Charles Shaw-Smith, Sarah E Flanagan, et al.
World Neurosurgery|February 7, 2016
Flow-Related Aneurysm within Glioblastoma: A Case Report and Review of LiteratureChibawanye I Ene, John D Nerva, Lynn McGrath, et al.
Pediatric and Developmental Pathology : the Official Journal of the Society for Pediatric Pathology and the Paediatric Pathology Society|March 22, 2018
Focal Congenital Hyperinsulinism as a Cause for Sudden Infant DeathAmish Chinoy, Indraneel Banerjee, Sarah E Flanagan, et al.
The Journal of Trauma|May 1, 1989
A revision of the Trauma ScoreH R Champion, W J Sacco, W S Copes, et al.
Diabetes, Obesity & Metabolism|October 9, 2007
Functional analysis of two Kir6.2 (KCNJ11) mutations, K170T and E322K, causing neonatal diabetesA I Tarasov, C A Girard, B Larkin, et al.
Diabetes & Metabolism|May 4, 2013
A novel GATA6 mutation leading to congenital heart defects and permanent neonatal diabetes: a case reportG Catli, A Abaci, S E Flanagan, et al.
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