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American Journal of Physiology. Endocrinology and Metabolism|March 11, 2000
Hepatic alpha- and beta-adrenergic receptors are not essential for the increase in R(a) during exercise in diabetesR H Coker, D B Lacy, P E Williams, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|May 8, 1999
Splanchnic glucagon kinetics in exercising alloxan-diabetic dogsR H Coker, D B Lacy, M G Krishna, et al.
American Journal of Physiology. Endocrinology and Metabolism|May 15, 2001
Stimulation of splanchnic glucose production during exercise in humans contains a glucagon-independent componentR H Coker, L Simonsen, J Bülow, et al.
The American Journal of Physiology|June 11, 1999
Prior exercise increases net hepatic glucose uptake during a glucose loadP Galassetti, R H Coker, D B Lacy, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|April 1, 1997
Sympathetic drive to liver and nonhepatic splanchnic tissue during heavy exerciseR H Coker, M G Krishna, D B Lacy, et al.
The American Journal of Physiology|December 31, 1997
Role of hepatic alpha- and beta-adrenergic receptor stimulation on hepatic glucose production during heavy exerciseR H Coker, M G Krishna, D B Lacy, et al.
The American Journal of Physiology|December 22, 1999
Role of a negative arterial-portal venous glucose gradient in the postexercise stateP Galassetti, Y Koyama, R H Coker, et al.
The American Journal of Physiology|December 22, 1999
Pancreatic innervation is not essential for exercise-induced changes in glucagon and insulin or glucose kineticsR H Coker, Y Koyama, D B Lacy, et al.
American Journal of Physiology. Endocrinology and Metabolism|August 19, 2000
Glucagon response to exercise is critical for accelerated hepatic glutamine metabolism and nitrogen disposalM G Krishna, R H Coker, D B Lacy, et al.
Metabolism: Clinical and Experimental|November 15, 1997
Sympathetic drive to liver and nonhepatic splanchnic tissue during prolonged exercise is increased in diabetesR H Coker, M G Krishna, B A Zinker, et al.
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