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R H Perry

Showing results (121-130 of 219) with videos related to

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Progress in Brain Research|January 1, 1986
The current status of the cortical cholinergic system in Alzheimer's disease and Parkinson's diseaseJ M Candy, E K Perry, R H Perry, et al.
Neuroscience Letters|December 13, 1982
Extensive loss of choline acetyltransferase activity is not reflected by neuronal loss in the nucleus of Meynert in Alzheimer's diseaseR H Perry, J M Candy, E K Perry, et al.
Acta Neurologica Scandinavica|May 1, 1997
Striatal dopaminergic loss without parkinsonism in a case of corticobasal degenerationE F Marshall, R H Perry, E K Perry, et al.
Journal of the Neurological Sciences|May 1, 1983
Pathological changes in the nucleus of Meynert in Alzheimer's and Parkinson's diseasesJ M Candy, R H Perry, E K Perry, et al.
Neuroreport|January 14, 1998
No association between a polymorphism in the presenilin 1 gene and dementia with Lewy bodiesA B Singleton, H Lamb, A Leake, et al.
Dementia (Basel, Switzerland)|September 1, 1994
Senile dementia of Lewy body type and Alzheimer type are biochemically distinct in terms of paired helical filaments and hyperphosphorylated tau proteinC R Harrington, R H Perry, E K Perry, et al.
Lancet (London, England)|June 18, 1994
Apolipoprotein E genes in Lewy body and Parkinson's diseaseR Benjamin, A Leake, J A Edwardson, et al.
Biological Psychiatry|February 15, 1991
Neocortical concentrations of neuropeptides in senile dementia of the Alzheimer and Lewy body type: comparison with Parkinson's disease and severity correlationsA Leake, E K Perry, R H Perry, et al.
Neurodegeneration : a Journal for Neurodegenerative Disorders, Neuroprotection, and Neuroregeneration|December 1, 1995
Effects of apolipoprotein E genotype on cortical neuropathology in senile dementia of the Lewy body and Alzheimer's diseaseR Benjamin, A Leake, P G Ince, et al.
Neuroscience Letters|September 30, 1983
Molecular forms of acetylcholinesterase in senile dementia of Alzheimer type: selective loss of the intermediate (10S) formJ R Atack, E K Perry, J R Bonham, et al.
Pageof 22

Showing results (121-130 of 219) with videos related to

Sort By:
Pageof 22
Progress in Brain Research|January 1, 1986
The current status of the cortical cholinergic system in Alzheimer's disease and Parkinson's diseaseJ M Candy, E K Perry, R H Perry, et al.
Neuroscience Letters|December 13, 1982
Extensive loss of choline acetyltransferase activity is not reflected by neuronal loss in the nucleus of Meynert in Alzheimer's diseaseR H Perry, J M Candy, E K Perry, et al.
Acta Neurologica Scandinavica|May 1, 1997
Striatal dopaminergic loss without parkinsonism in a case of corticobasal degenerationE F Marshall, R H Perry, E K Perry, et al.
Journal of the Neurological Sciences|May 1, 1983
Pathological changes in the nucleus of Meynert in Alzheimer's and Parkinson's diseasesJ M Candy, R H Perry, E K Perry, et al.
Neuroreport|January 14, 1998
No association between a polymorphism in the presenilin 1 gene and dementia with Lewy bodiesA B Singleton, H Lamb, A Leake, et al.
Dementia (Basel, Switzerland)|September 1, 1994
Senile dementia of Lewy body type and Alzheimer type are biochemically distinct in terms of paired helical filaments and hyperphosphorylated tau proteinC R Harrington, R H Perry, E K Perry, et al.
Lancet (London, England)|June 18, 1994
Apolipoprotein E genes in Lewy body and Parkinson's diseaseR Benjamin, A Leake, J A Edwardson, et al.
Biological Psychiatry|February 15, 1991
Neocortical concentrations of neuropeptides in senile dementia of the Alzheimer and Lewy body type: comparison with Parkinson's disease and severity correlationsA Leake, E K Perry, R H Perry, et al.
Neurodegeneration : a Journal for Neurodegenerative Disorders, Neuroprotection, and Neuroregeneration|December 1, 1995
Effects of apolipoprotein E genotype on cortical neuropathology in senile dementia of the Lewy body and Alzheimer's diseaseR Benjamin, A Leake, P G Ince, et al.
Neuroscience Letters|September 30, 1983
Molecular forms of acetylcholinesterase in senile dementia of Alzheimer type: selective loss of the intermediate (10S) formJ R Atack, E K Perry, J R Bonham, et al.
Pageof 22