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R Higgs

Showing results (271-280 of 469) with videos related to

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Experimental Hematology|January 14, 2018
Phenotypic and molecular characterization of a serum-free miniature erythroid differentiation system suitable for high-throughput screening and single-cell assaysSachith Mettananda, Kevin Clark, Chris A Fisher, et al.
Human Molecular Genetics|December 1, 1996
ATRX encodes a novel member of the SNF2 family of proteins: mutations point to a common mechanism underlying the ATR-X syndromeD J Picketts, D R Higgs, S Bachoo, et al.
The New England Journal of Medicine|July 21, 1983
alpha-Thalassemia reduces the hemolytic rate in homozygous sickle-cell diseaseK de Ceulaer, D R Higgs, D J Weatherall, et al.
Genome Research|April 3, 2004
Comparative analysis of the alpha-like globin clusters in mouse, rat, and human chromosomes indicates a mechanism underlying breaks in conserved syntenyCristina Tufarelli, Ross Hardison, Webb Miller, et al.
British Journal of Haematology|January 1, 1981
The molecular basis for the haemoglobin Bart's hydrops fetalis syndrome in CyprusT Sophocleous, D R Higgs, B Aldridge, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 1, 1990
Inactivation of human alpha-globin gene expression by a de novo deletion located upstream of the alpha-globin gene clusterS A Liebhaber, E U Griese, I Weiss, et al.
Genome Research|September 15, 2017
Sasquatch: predicting the impact of regulatory SNPs on transcription factor binding from cell- and tissue-specific DNase footprintsRon Schwessinger, Maria C Suciu, Simon J McGowan, et al.
Blood|April 1, 1989
A review of the molecular genetics of the human alpha-globin gene clusterD R Higgs, M A Vickers, A O Wilkie, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 21, 2008
Neuronal death resulting from targeted disruption of the Snf2 protein ATRX is mediated by p53Claudia Seah, Michael A Levy, Yan Jiang, et al.
The Journal of Clinical Investigation|January 1, 1987
Different hematologic phenotypes are associated with the leftward (-alpha 4.2) and rightward (-alpha 3.7) alpha+-thalassemia deletionsD K Bowden, A V Hill, D R Higgs, et al.
Pageof 47

Showing results (271-280 of 469) with videos related to

Sort By:
Pageof 47
Experimental Hematology|January 14, 2018
Phenotypic and molecular characterization of a serum-free miniature erythroid differentiation system suitable for high-throughput screening and single-cell assaysSachith Mettananda, Kevin Clark, Chris A Fisher, et al.
Human Molecular Genetics|December 1, 1996
ATRX encodes a novel member of the SNF2 family of proteins: mutations point to a common mechanism underlying the ATR-X syndromeD J Picketts, D R Higgs, S Bachoo, et al.
The New England Journal of Medicine|July 21, 1983
alpha-Thalassemia reduces the hemolytic rate in homozygous sickle-cell diseaseK de Ceulaer, D R Higgs, D J Weatherall, et al.
Genome Research|April 3, 2004
Comparative analysis of the alpha-like globin clusters in mouse, rat, and human chromosomes indicates a mechanism underlying breaks in conserved syntenyCristina Tufarelli, Ross Hardison, Webb Miller, et al.
British Journal of Haematology|January 1, 1981
The molecular basis for the haemoglobin Bart's hydrops fetalis syndrome in CyprusT Sophocleous, D R Higgs, B Aldridge, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 1, 1990
Inactivation of human alpha-globin gene expression by a de novo deletion located upstream of the alpha-globin gene clusterS A Liebhaber, E U Griese, I Weiss, et al.
Genome Research|September 15, 2017
Sasquatch: predicting the impact of regulatory SNPs on transcription factor binding from cell- and tissue-specific DNase footprintsRon Schwessinger, Maria C Suciu, Simon J McGowan, et al.
Blood|April 1, 1989
A review of the molecular genetics of the human alpha-globin gene clusterD R Higgs, M A Vickers, A O Wilkie, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 21, 2008
Neuronal death resulting from targeted disruption of the Snf2 protein ATRX is mediated by p53Claudia Seah, Michael A Levy, Yan Jiang, et al.
The Journal of Clinical Investigation|January 1, 1987
Different hematologic phenotypes are associated with the leftward (-alpha 4.2) and rightward (-alpha 3.7) alpha+-thalassemia deletionsD K Bowden, A V Hill, D R Higgs, et al.
Pageof 47