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Experimental Cell Research|May 23, 1998
CaMK-II inhibition reduces cyclin D1 levels and enhances the association of p27kip1 with Cdk2 to cause G1 arrest in NIH 3T3 cellsT A Morris, R J DeLorenzo, R M TombesBrain Research|May 31, 2001
Long-term alteration of calcium homeostatic mechanisms in the pilocarpine model of temporal lobe epilepsyM Raza, S Pal, A Rafiq, et al.Brain Research|July 17, 1999
Global ischemia-induced inhibition of the coupling ratio of calcium uptake and ATP hydrolysis by rat whole brain microsomal Mg(2+)/Ca(2+) ATPaseJ T Parsons, S B Churn, R J DeLorenzoAdvances in Experimental Medicine and Biology|January 1, 1987
Molecular mechanisms of neuronal excitability: possible involvement of CaM kinase II in seizure activityW C Taft, J R Goldenring, R J DeLorenzoJournal of Neurochemistry|December 1, 1984
Tubulin-associated calmodulin-dependent kinase: evidence for an endogenous complex of tubulin with a calcium-calmodulin-dependent kinaseJ R Goldenring, J E Casanova, R J DeLorenzoBrain Research|June 12, 1992
Excitatory amino acid receptor activation produces a selective and long-lasting modulation of gene expression in hippocampal neuronsE R Jakoi, S Sombati, C Gerwin, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 1, 1995
Excitotoxic activation of the NMDA receptor results in inhibition of calcium/calmodulin kinase II activity in cultured hippocampal neuronsS B Churn, D Limbrick, S Sombati, et al.Cell Calcium|October 6, 2000
Induction of spontaneous recurrent epileptiform discharges causes long-term changes in intracellular calcium homeostatic mechanismsS Pal, D D Limbrick, A Rafiq, et al.Brain Research|January 22, 2000
In vitro status epilepticus causes sustained elevation of intracellular calcium levels in hippocampal neuronsS Pal, S Sombati, D D Limbrick, et al.Stroke|February 1, 1993
Excitotoxicity affects membrane potential and calmodulin kinase II activity in cultured rat cortical neuronsS B Churn, S Sombati, W C Taft, et al.Pageof 14