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Human Gene Therapy|August 1, 1995
Fluorescence-based selection of retrovirally transduced cells in the absence of a marker gene: direct selection of transduced type B Niemann-Pick disease cells and evidence for bystander correctionP L Yeyati, V Agmon, C Fillat, et al.Molecular Medicine (Cambridge, Mass.)|February 10, 2000
Twenty novel mutations in the alpha-galactosidase A gene causing Fabry diseaseA K Topaloglu, G A Ashley, B Tong, et al.The Tohoku Journal of Experimental Medicine|October 1, 1995
Identification and expression of a missense mutation (Y446C) in the acid sphingomyelinase gene from a Japanese patient with type A Niemann-Pick diseaseT Takahashi, M Suchi, W Sato, et al.Muscle & Nerve|March 1, 1988
Hexosaminidase A activity and amyotrophic lateral sclerosisM Gudesblatt, M D Ludman, J A Cohen, et al.The Biochemical Journal|April 16, 1998
Presence of dopa and amino acid hydroperoxides in proteins modified with advanced glycation end products (AGEs): amino acid oxidation products as a possible source of oxidative stress induced by AGE proteinsS Fu, M X Fu, J W Baynes, et al.Journal of Nuclear Medicine : Official Publication, Society of Nuclear Medicine|September 18, 1997
Manipulation of blood clearance to optimize delivery of residualizing label-antibody conjugates to tumor cells in vivoR Stein, D M Goldenberg, G L Ong, et al.Biochemistry|August 29, 1995
N epsilon-(carboxymethyl)lysine is a dominant advanced glycation end product (AGE) antigen in tissue proteinsS Reddy, J Bichler, K J Wells-Knecht, et al.International Journal of Radiation Applications and Instrumentation. Part B, Nuclear Medicine and Biology|April 1, 1992
Quantitative assessment of lipoprotein metabolism by positron emission tomography with an 18F-containing residualizing labelA Daugherty, M R Kilbourn, C S Dence, et al.American Journal of Human Genetics|July 1, 1979
Argininosuccinic aciduria: prenatal studies in a family at riskL D Fleisher, D K Rassin, R J Desnick, et al.Molecular Genetics and Metabolism|June 5, 1999
Identification and characterization of the molecular lesion causing mucopolysaccharidosis type I in catsX He, C M Li, C M Simonaro, et al.Pageof 40