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Biochemical Pharmacology|June 1, 1989
Blockade by lipoxygenase inhibitors of Ca2+-dependent insulin secretion from permeabilized rat islets. A molecular mechanism distinct from that of alpha 2-adrenergic agonistsS A MetzDiabetes|January 1, 1988
Epinephrine impairs insulin release by a mechanism distal to calcium mobilization. Similarity to lipoxygenase inhibitorsS A MetzDiabetes|December 1, 1991
The pancreatic islet as Rubik's Cube. Is phospholipid hydrolysis a piece of the puzzle?S A MetzLife Sciences|June 9, 1986
A re-interpretation of lipoxygenase-dependent insulin release: which metabolites of arachidonic acid, or none?S A MetzProstaglandins|January 1, 1984
Is phospholipase A2 a "glucose sensor" responsible for the phasic pattern of insulin release?S A MetzThe American Journal of Medicine|November 28, 1988
Membrane phospholipid turnover as an intermediary step in insulin secretion. Putative roles of phospholipases in cell signalingS A MetzProstaglandins and Medicine|December 1, 1981
Feedback modulation of glucose-induced insulin secretion by arachidonic acid metabolites: possible molecular mechanisms and relevance to diabetes mellitusS A MetzBiochemical Pharmacology|June 1, 1988
Dantrolene-induced inhibition of insulin release. A mechanism independent of effects on calcium fluxesS A MetzThe Journal of Pharmacology and Experimental Therapeutics|September 1, 1986
p-Hydroxymercuribenzoic acid inhibits arachidonic acid esterification into two distinct pools and stimulates insulin release in intact rat isletsS A MetzThe Biochemical Journal|February 1, 1987
Metabolism of lysophospholipids in intact rat islets. The insulin secretagogue p-hydroxymercuribenzoic acid impairs lysophosphatidylcholine catabolism and permits its accumulationS A MetzPageof 19