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Biochemical and Biophysical Research Communications|May 30, 1989
Retention of specific protein kinase C isozymes following chronic phorbol ester treatment in BC3H-1 myocytesD R Cooper, J E Watson, M Acevedo-Duncan, et al.
The Biochemical Journal|August 1, 1989
Insulin-like effects of epidermal growth factor and insulin-like growth factor-I on [3H]2-deoxyglucose uptake, diacylglycerol generation and protein kinase C activation in BC3H-1 myocytesR V Farese, G P Nair, C G Sierra, et al.
Diabetes|November 1, 1990
Sulfonylurea-stimulated glucose transport association with diacylglycerollike activation of protein kinase C in BC3H1 myocytesD R Cooper, M C Vila, J E Watson, et al.
Endocrinology|May 1, 1984
Interaction of insulin-like growth factors with a nonfusing mouse muscle cell line: binding, action, and receptor down-regulationM A de Vroede, J A Romanus, M L Standaert, et al.
Metabolism: Clinical and Experimental|November 1, 1990
Protein kinase C inhibitors block insulin and PMA-stimulated hexose transport in isolated rat adipocytes and BC3H-1 myocytesM L Standaert, D J Buckley, T Ishizuka, et al.
The Biochemical Journal|November 15, 1988
Mechanisms whereby insulin increases diacylglycerol in BC3H-1 myocytesR V Farese, D R Cooper, T S Konda, et al.
FEBS Letters|April 6, 1987
Insulin but not phorbol ester treatment increases phosphorylation of vinculin by protein kinase C in BC3H-1 myocytesD R Cooper, C M de Ruiz Galaretta, L F Fanjul, et al.
The Biochemical Journal|October 15, 1985
The de novo phospholipid effect of insulin is associated with increases in diacylglycerol, but not inositol phosphates or cytosolic Ca2+R V Farese, J S Davis, D E Barnes, et al.
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