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R John Solaro

Showing results (111-120 of 200) with videos related to

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Scientific Reports|October 28, 2021
Negative inotropic mechanisms of β-cardiotoxin in cardiomyocytes by depression of myofilament ATPase activity without activation of the classical β-adrenergic pathwayTuchakorn Lertwanakarn, Montamas Suntravat, Elda E Sánchez, et al.
The Journal of Biological Chemistry|September 11, 2004
Cardiac troponin T isoforms affect the Ca(2+) sensitivity of force development in the presence of slow skeletal troponin I: insights into the role of troponin T isoforms in the fetal heartAldrin V Gomes, Gayathri Venkatraman, Jonathan P Davis, et al.
Basic Research in Cardiology|January 29, 2010
PDE5A suppression of acute beta-adrenergic activation requires modulation of myocyte beta-3 signaling coupled to PKG-mediated troponin I phosphorylationDong I Lee, Susan Vahebi, Carlo Gabriele Tocchetti, et al.
Journal of Muscle Research and Cell Motility|April 25, 2012
Protein kinase C depresses cardiac myocyte power output and attenuates myofilament responses induced by protein kinase AAaron C Hinken, Laurin M Hanft, Sarah B Scruggs, et al.
Molecular & Cellular Proteomics : MCP|May 7, 2010
A novel, in-solution separation of endogenous cardiac sarcomeric proteins and identification of distinct charged variants of regulatory light chainSarah B Scruggs, Rick Reisdorph, Mike L Armstrong, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|May 1, 2023
A perspective on Notch signalling in progression and arrhythmogenesis in familial hypertrophic and dilated cardiomyopathiesPaulina Langa, Sanam Shafaattalab, Paul H Goldspink, et al.
Archives of Biochemistry and Biophysics|October 23, 2012
Post-translational modifications of myofilament proteins involved in length-dependent prolongation of relaxation in rabbit right ventricular myocardiumMichelle M Monasky, Domenico M Taglieri, Alice K Jacobson, et al.
Journal of Molecular and Cellular Cardiology|February 1, 2006
Partial replacement of cardiac troponin I with a non-phosphorylatable mutant at serines 43/45 attenuates the contractile dysfunction associated with PKCepsilon phosphorylationSarah B Scruggs, Lori A Walker, Theodore Lyu, et al.
Frontiers in Physiology|April 9, 2015
Novel insights into mechanisms for Pak1-mediated regulation of cardiac Ca(2+) homeostasisYanwen Wang, Hoyee Tsui, Emma L Bolton, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 3, 2016
β-Arrestin mediates the Frank-Starling mechanism of cardiac contractilityDennis M Abraham, Robert T Davis, Chad M Warren, et al.
Pageof 20

Showing results (111-120 of 200) with videos related to

Sort By:
Pageof 20
Scientific Reports|October 28, 2021
Negative inotropic mechanisms of β-cardiotoxin in cardiomyocytes by depression of myofilament ATPase activity without activation of the classical β-adrenergic pathwayTuchakorn Lertwanakarn, Montamas Suntravat, Elda E Sánchez, et al.
The Journal of Biological Chemistry|September 11, 2004
Cardiac troponin T isoforms affect the Ca(2+) sensitivity of force development in the presence of slow skeletal troponin I: insights into the role of troponin T isoforms in the fetal heartAldrin V Gomes, Gayathri Venkatraman, Jonathan P Davis, et al.
Basic Research in Cardiology|January 29, 2010
PDE5A suppression of acute beta-adrenergic activation requires modulation of myocyte beta-3 signaling coupled to PKG-mediated troponin I phosphorylationDong I Lee, Susan Vahebi, Carlo Gabriele Tocchetti, et al.
Journal of Muscle Research and Cell Motility|April 25, 2012
Protein kinase C depresses cardiac myocyte power output and attenuates myofilament responses induced by protein kinase AAaron C Hinken, Laurin M Hanft, Sarah B Scruggs, et al.
Molecular & Cellular Proteomics : MCP|May 7, 2010
A novel, in-solution separation of endogenous cardiac sarcomeric proteins and identification of distinct charged variants of regulatory light chainSarah B Scruggs, Rick Reisdorph, Mike L Armstrong, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|May 1, 2023
A perspective on Notch signalling in progression and arrhythmogenesis in familial hypertrophic and dilated cardiomyopathiesPaulina Langa, Sanam Shafaattalab, Paul H Goldspink, et al.
Archives of Biochemistry and Biophysics|October 23, 2012
Post-translational modifications of myofilament proteins involved in length-dependent prolongation of relaxation in rabbit right ventricular myocardiumMichelle M Monasky, Domenico M Taglieri, Alice K Jacobson, et al.
Journal of Molecular and Cellular Cardiology|February 1, 2006
Partial replacement of cardiac troponin I with a non-phosphorylatable mutant at serines 43/45 attenuates the contractile dysfunction associated with PKCepsilon phosphorylationSarah B Scruggs, Lori A Walker, Theodore Lyu, et al.
Frontiers in Physiology|April 9, 2015
Novel insights into mechanisms for Pak1-mediated regulation of cardiac Ca(2+) homeostasisYanwen Wang, Hoyee Tsui, Emma L Bolton, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 3, 2016
β-Arrestin mediates the Frank-Starling mechanism of cardiac contractilityDennis M Abraham, Robert T Davis, Chad M Warren, et al.
Pageof 20