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American Journal of Physiology. Heart and Circulatory Physiology|July 4, 2006
Left ventricular myofilament dysfunction in rat experimental hypertrophy and congestive heart failureRashad J Belin, Marius P Sumandea, Tomoyoshi Kobayashi, et al.Archives of Biochemistry and Biophysics|December 13, 2012
Tropomyosin Ser-283 pseudo-phosphorylation slows myofibril relaxationBenjamin R Nixon, Bin Liu, Beatrice Scellini, et al.Circulation. Arrhythmia and Electrophysiology|September 14, 2014
Pak1 is required to maintain ventricular Ca²⁺ homeostasis and electrophysiological stability through SERCA2a regulation in miceYanwen Wang, Hoyee Tsui, Yunbo Ke, et al.The Journal of Biological Chemistry|November 14, 2012
Tropomyosin dephosphorylation results in compensated cardiac hypertrophyEmily M Schulz, Richard N Correll, Hajer N Sheikh, et al.Journal of Molecular and Cellular Cardiology|September 22, 2010
Neonatal gene transfer of Serca2a delays onset of hypertrophic remodeling and improves function in familial hypertrophic cardiomyopathyJames R Peña, Ariani C Szkudlarek, Chad M Warren, et al.Circulation. Heart Failure|November 6, 2019
Sphingosine-1-Phosphate Receptor Modulator, FTY720, Improves Diastolic Dysfunction and Partially Reverses Atrial Remodeling in a Tm-E180G Mouse Model Linked to Hypertrophic CardiomyopathyDavid M Ryba, Chad M Warren, Chehade N Karam, et al.Circulation|November 16, 2011
Pak1 as a novel therapeutic target for antihypertrophic treatment in the heartWei Liu, Min Zi, Ronald Naumann, et al.Circulation Research|February 21, 2012
Ranolazine improves cardiac diastolic dysfunction through modulation of myofilament calcium sensitivityJoshua D Lovelock, Michelle M Monasky, Euy-Myoung Jeong, et al.American Journal of Physiology. Heart and Circulatory Physiology|May 12, 2009
An internal domain of beta-tropomyosin increases myofilament Ca(2+) sensitivityGanapathy Jagatheesan, Sudarsan Rajan, Emily M Schulz, et al.American Journal of Physiology. Heart and Circulatory Physiology|June 28, 2011
Activation of Pak1/Akt/eNOS signaling following sphingosine-1-phosphate release as part of a mechanism protecting cardiomyocytes against ischemic cell injuryEmmanuel Eroume A Egom, Tamer M A Mohamed, Mamas A Mamas, et al.Pageof 20