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R Jove

Showing results (41-50 of 90) with videos related to

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Molecular and Cellular Biology|January 1, 1995
Raf-1 N-terminal sequences necessary for Ras-Raf interaction and signal transductionK Pumiglia, Y H Chow, J Fabian, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|April 1, 1998
Elevated c-Src protein expression is an early event in colonic neoplasiaS Iravani, W Mao, L Fu, et al.
The Journal of Biological Chemistry|June 16, 1995
The Ras GTPase-activating protein (GAP) is an SH3 domain-binding protein and substrate for the Src-related tyrosine kinase, HckS D Briggs, S S Bryant, R Jove, et al.
Molecular and Cellular Biology|May 5, 1998
Stat3 activation by Src induces specific gene regulation and is required for cell transformationJ Turkson, T Bowman, R Garcia, et al.
Cancer Control : Journal of the Moffitt Cancer Center|January 25, 2002
Prostate adenocarcinoma: cellular and molecular abnormalitiesL B Mora, R Buettner, N Ahmad, et al.
The Journal of Biological Chemistry|March 1, 1996
N-terminal sequences contained in the Src homology 2 and 3 domains of p120 GTPase-activating protein are required for full catalytic activity toward RasS S Bryant, A L Mitchell, F Collins, et al.
Cancer Research|March 1, 1997
Role of epidermal growth factor receptor and STAT-3 activation in autonomous proliferation of SUM-102PT human breast cancer cellsC I Sartor, M L Dziubinski, C L Yu, et al.
Cell|July 1, 1984
Splicing pathways of SV40 mRNAs in X. laevis oocytes differ in their requirements for snRNPsA Fradin, R Jove, C Hemenway, et al.
Journal of Virology|December 1, 1986
Genetic lesions involved in temperature sensitivity of the src gene products of four Rous sarcoma virus mutantsB J Mayer, R Jove, J F Krane, et al.
Oncogene|May 18, 2000
Activation of Stat3 preassembled with platelet-derived growth factor beta receptors requires Src kinase activityY Z Wang, W Wharton, R Garcia, et al.
Pageof 9

Showing results (41-50 of 90) with videos related to

Sort By:
Pageof 9
Molecular and Cellular Biology|January 1, 1995
Raf-1 N-terminal sequences necessary for Ras-Raf interaction and signal transductionK Pumiglia, Y H Chow, J Fabian, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|April 1, 1998
Elevated c-Src protein expression is an early event in colonic neoplasiaS Iravani, W Mao, L Fu, et al.
The Journal of Biological Chemistry|June 16, 1995
The Ras GTPase-activating protein (GAP) is an SH3 domain-binding protein and substrate for the Src-related tyrosine kinase, HckS D Briggs, S S Bryant, R Jove, et al.
Molecular and Cellular Biology|May 5, 1998
Stat3 activation by Src induces specific gene regulation and is required for cell transformationJ Turkson, T Bowman, R Garcia, et al.
Cancer Control : Journal of the Moffitt Cancer Center|January 25, 2002
Prostate adenocarcinoma: cellular and molecular abnormalitiesL B Mora, R Buettner, N Ahmad, et al.
The Journal of Biological Chemistry|March 1, 1996
N-terminal sequences contained in the Src homology 2 and 3 domains of p120 GTPase-activating protein are required for full catalytic activity toward RasS S Bryant, A L Mitchell, F Collins, et al.
Cancer Research|March 1, 1997
Role of epidermal growth factor receptor and STAT-3 activation in autonomous proliferation of SUM-102PT human breast cancer cellsC I Sartor, M L Dziubinski, C L Yu, et al.
Cell|July 1, 1984
Splicing pathways of SV40 mRNAs in X. laevis oocytes differ in their requirements for snRNPsA Fradin, R Jove, C Hemenway, et al.
Journal of Virology|December 1, 1986
Genetic lesions involved in temperature sensitivity of the src gene products of four Rous sarcoma virus mutantsB J Mayer, R Jove, J F Krane, et al.
Oncogene|May 18, 2000
Activation of Stat3 preassembled with platelet-derived growth factor beta receptors requires Src kinase activityY Z Wang, W Wharton, R Garcia, et al.
Pageof 9